Weinstein IB - Search Results

1

Induction of apoptosis by the garlic-derived compound S-allylmercaptocysteine (SAMC) is associated with microtubule depolymerization and c-Jun NH(2)-terminal kinase 1 activation.

Xiao D, Pinto JT, Soh JW, Deguchi A, Gundersen GG, Palazzo AF, Yoon JT, Shirin H, Weinstein IB. Xiao D, et al. Among authors: weinstein ib. Cancer Res. 2003 Oct 15;63(20):6825-37. Cancer Res. 2003. PMID: 14583480

2

CP248, a derivative of exisulind, causes growth inhibition, mitotic arrest, and abnormalities in microtubule polymerization in glioma cells.

Yoon JT, Palazzo AF, Xiao D, Delohery TM, Warburton PE, Bruce JN, Thompson WJ, Sperl G, Whitehead C, Fetter J, Pamukcu R, Gundersen GG, Weinstein IB. Yoon JT, et al. Among authors: weinstein ib. Mol Cancer Ther. 2002 Apr;1(6):393-404. Mol Cancer Ther. 2002. PMID: 12477052

3

Vasodilator-stimulated phosphoprotein (VASP) phosphorylation provides a biomarker for the action of exisulind and related agents that activate protein kinase G.

Deguchi A, Soh JW, Li H, Pamukcu R, Thompson WJ, Weinstein IB. Deguchi A, et al. Among authors: weinstein ib. Mol Cancer Ther. 2002 Aug;1(10):803-9. Mol Cancer Ther. 2002. PMID: 12492113

4

Activation of protein kinase G is sufficient to induce apoptosis and inhibit cell migration in colon cancer cells.

Deguchi A, Thompson WJ, Weinstein IB. Deguchi A, et al. Among authors: weinstein ib. Cancer Res. 2004 Jun 1;64(11):3966-73. doi: 10.1158/0008-5472.CAN-03-3740. Cancer Res. 2004. PMID: 15173009

5

(-)-Epigallocatechin gallate and polyphenon E inhibit growth and activation of the epidermal growth factor receptor and human epidermal growth factor receptor-2 signaling pathways in human colon cancer cells.

Shimizu M, Deguchi A, Lim JT, Moriwaki H, Kopelovich L, Weinstein IB. Shimizu M, et al. Among authors: weinstein ib. Clin Cancer Res. 2005 Apr 1;11(7):2735-46. doi: 10.1158/1078-0432.CCR-04-2014. Clin Cancer Res. 2005. PMID: 15814656

6

Activation of protein kinase G up-regulates expression of 15-lipoxygenase-1 in human colon cancer cells.

Deguchi A, Xing SW, Shureiqi I, Yang P, Newman RA, Lippman SM, Feinmark SJ, Oehlen B, Weinstein IB. Deguchi A, et al. Among authors: weinstein ib. Cancer Res. 2005 Sep 15;65(18):8442-7. doi: 10.1158/0008-5472.CAN-05-1109. Cancer Res. 2005. PMID: 16166323

7

Effects of a series of organosulfur compounds on mitotic arrest and induction of apoptosis in colon cancer cells.

Xiao D, Pinto JT, Gundersen GG, Weinstein IB. Xiao D, et al. Among authors: weinstein ib. Mol Cancer Ther. 2005 Sep;4(9):1388-98. doi: 10.1158/1535-7163.MCT-05-0152. Mol Cancer Ther. 2005. PMID: 16170031

8

The sulindac derivatives OSI-461, OSIP486823, and OSIP487703 arrest colon cancer cells in mitosis by causing microtubule depolymerization.

Xiao D, Deguchi A, Gundersen GG, Oehlen B, Arnold L, Weinstein IB. Xiao D, et al. Among authors: weinstein ib. Mol Cancer Ther. 2006 Jan;5(1):60-7. doi: 10.1158/1535-7163.MCT-05-0260. Mol Cancer Ther. 2006. PMID: 16432163

9

Activation of protein kinase G Increases the expression of p21CIP1, p27KIP1, and histidine triad protein 1 through Sp1.

Cen B, Deguchi A, Weinstein IB. Cen B, et al. Among authors: weinstein ib. Cancer Res. 2008 Jul 1;68(13):5355-62. doi: 10.1158/0008-5472.CAN-07-6869. Cancer Res. 2008. PMID: 18593937

10

Cyclic GMP mediates apoptosis induced by sulindac derivatives via activation of c-Jun NH2-terminal kinase 1.

Soh JW, Mao Y, Kim MG, Pamukcu R, Li H, Piazza GA, Thompson WJ, Weinstein IB. Soh JW, et al. Among authors: weinstein ib. Clin Cancer Res. 2000 Oct;6(10):4136-41. Clin Cancer Res. 2000. PMID: 11051267

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