Li RA - Search Results

1

Mitochondrial ATP-sensitive potassium channel activation protects cerebellar granule neurons from apoptosis induced by oxidative stress.

Teshima Y, Akao M, Li RA, Chong TH, Baumgartner WA, Johnston MV, Marbán E. Teshima Y, et al. Among authors: li ra. Stroke. 2003 Jul;34(7):1796-802. doi: 10.1161/01.STR.0000077017.60947.AE. Epub 2003 Jun 5. Stroke. 2003. PMID: 12791941

2

Dominant-negative suppression of HCN1- and HCN2-encoded pacemaker currents by an engineered HCN1 construct: insights into structure-function relationships and multimerization.

Xue T, Marbán E, Li RA. Xue T, et al. Among authors: li ra. Circ Res. 2002 Jun 28;90(12):1267-73. doi: 10.1161/01.res.0000024390.97889.c6. Circ Res. 2002. PMID: 12089064

3

Identification of a surface charged residue in the S3-S4 linker of the pacemaker (HCN) channel that influences activation gating.

Henrikson CA, Xue T, Dong P, Sang D, Marban E, Li RA. Henrikson CA, et al. Among authors: li ra. J Biol Chem. 2003 Apr 18;278(16):13647-54. doi: 10.1074/jbc.M211025200. Epub 2003 Feb 11. J Biol Chem. 2003. PMID: 12582169 Free article.

4

Dependence of mu-conotoxin block of sodium channels on ionic strength but not on the permeating [Na+]: implications for the distinctive mechanistic interactions between Na+ and K+ channel pore-blocking toxins and their molecular targets.

Li RA, Hui K, French RJ, Sato K, Henrikson CA, Tomaselli GF, Marbán E. Li RA, et al. J Biol Chem. 2003 Aug 15;278(33):30912-9. doi: 10.1074/jbc.M301039200. Epub 2003 May 21. J Biol Chem. 2003. PMID: 12764145 Free article.

5

Molecular basis of electrocardiographic ST-segment elevation.

Li RA, Leppo M, Miki T, Seino S, Marbán E. Li RA, et al. Circ Res. 2000 Nov 10;87(10):837-9. doi: 10.1161/01.res.87.10.837. Circ Res. 2000. PMID: 11073877

6

Structural basis of differences in isoform-specific gating and lidocaine block between cardiac and skeletal muscle sodium channels.

Li RA, Ennis IL, Tomaselli GF, Marbán E. Li RA, et al. Mol Pharmacol. 2002 Jan;61(1):136-41. doi: 10.1124/mol.61.1.136. Mol Pharmacol. 2002. PMID: 11752214

7

Charge conversion enables quantification of the proximity between a normally-neutral mu-conotoxin (GIIIA) site and the Na+ channel pore.

Li RA, Sato K, Kodama K, Kohno T, Xue T, Tomaselli GF, Marbán E. Li RA, et al. FEBS Lett. 2002 Jan 30;511(1-3):159-64. doi: 10.1016/s0014-5793(01)03316-6. FEBS Lett. 2002. PMID: 11821068 Free article.

8

Non-equilibrium behavior of HCN channels: insights into the role of HCN channels in native and engineered pacemakers.

Azene EM, Xue T, Marbán E, Tomaselli GF, Li RA. Azene EM, et al. Among authors: li ra. Cardiovasc Res. 2005 Aug 1;67(2):263-73. doi: 10.1016/j.cardiores.2005.03.006. Epub 2005 Apr 21. Cardiovasc Res. 2005. PMID: 16005302

9

Molecular architecture of the voltage-dependent Na channel: functional evidence for alpha helices in the pore.

Yamagishi T, Li RA, Hsu K, Marbán E, Tomaselli GF. Yamagishi T, et al. Among authors: li ra. J Gen Physiol. 2001 Aug;118(2):171-82. doi: 10.1085/jgp.118.2.171. J Gen Physiol. 2001. PMID: 11479344 Free PMC article.

10

Charged residues between the selectivity filter and S6 segments contribute to the permeation phenotype of the sodium channel.

Li RA, Vélez P, Chiamvimonvat N, Tomaselli GF, Marbán E. Li RA, et al. J Gen Physiol. 2000 Jan;115(1):81-92. doi: 10.1085/jgp.115.1.81. J Gen Physiol. 2000. PMID: 10613920 Free PMC article.

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