Rosenblat M - Search Results

1

Paraoxonase (PON1) deficiency is associated with increased macrophage oxidative stress: studies in PON1-knockout mice.

Rozenberg O, Rosenblat M, Coleman R, Shih DM, Aviram M. Rozenberg O, et al. Among authors: rosenblat m. Free Radic Biol Med. 2003 Mar 15;34(6):774-84. doi: 10.1016/s0891-5849(02)01429-6. Free Radic Biol Med. 2003. PMID: 12633754

2

Oxysterol-induced activation of macrophage NADPH-oxidase enhances cell-mediated oxidation of LDL in the atherosclerotic apolipoprotein E deficient mouse: inhibitory role for vitamin E.

Rosenblat M, Aviram M. Rosenblat M, et al. Atherosclerosis. 2002 Jan;160(1):69-80. doi: 10.1016/s0021-9150(01)00563-9. Atherosclerosis. 2002. PMID: 11755924

3

Serum paraoxonase activity and the extent of lipid peroxidation are not affected by increased levels of human apolipoprotein A-I: studies in transgenic mice.

Rosenblat M, Grunfeld O, Hayek T, Aviram M. Rosenblat M, et al. Clin Chem Lab Med. 2002 Jan;40(1):9-14. doi: 10.1515/CCLM.2002.003. Clin Chem Lab Med. 2002. PMID: 11916277

4

Increased macrophage glutathione content reduces cell-mediated oxidation of LDL and atherosclerosis in apolipoprotein E-deficient mice.

Rosenblat M, Coleman R, Aviram M. Rosenblat M, et al. Atherosclerosis. 2002 Jul;163(1):17-28. doi: 10.1016/s0021-9150(01)00744-4. Atherosclerosis. 2002. PMID: 12048118

5

Mouse macrophage paraoxonase 2 activity is increased whereas cellular paraoxonase 3 activity is decreased under oxidative stress.

Rosenblat M, Draganov D, Watson CE, Bisgaier CL, La Du BN, Aviram M. Rosenblat M, et al. Arterioscler Thromb Vasc Biol. 2003 Mar 1;23(3):468-74. doi: 10.1161/01.ATV.0000059385.95664.4D. Epub 2003 Jan 30. Arterioscler Thromb Vasc Biol. 2003. PMID: 12615656

6

Decreased macrophage paraoxonase 2 expression in patients with hypercholesterolemia is the result of their increased cellular cholesterol content: effect of atorvastatin therapy.

Rosenblat M, Hayek T, Hussein K, Aviram M. Rosenblat M, et al. Arterioscler Thromb Vasc Biol. 2004 Jan;24(1):175-80. doi: 10.1161/01.ATV.0000104011.88939.06. Epub 2003 Oct 30. Arterioscler Thromb Vasc Biol. 2004. PMID: 14592851

7

Paraoxonases 1, 2, and 3, oxidative stress, and macrophage foam cell formation during atherosclerosis development.

Aviram M, Rosenblat M. Aviram M, et al. Among authors: rosenblat m. Free Radic Biol Med. 2004 Nov 1;37(9):1304-16. doi: 10.1016/j.freeradbiomed.2004.06.030. Free Radic Biol Med. 2004. PMID: 15454271 Review.

8

Paraoxonase 1 (PON1) enhances HDL-mediated macrophage cholesterol efflux via the ABCA1 transporter in association with increased HDL binding to the cells: a possible role for lysophosphatidylcholine.

Rosenblat M, Vaya J, Shih D, Aviram M. Rosenblat M, et al. Atherosclerosis. 2005 Mar;179(1):69-77. doi: 10.1016/j.atherosclerosis.2004.10.028. Epub 2004 Dec 29. Atherosclerosis. 2005. PMID: 15721011

9

Paraoxonase 1 (PON1) is a more potent antioxidant and stimulant of macrophage cholesterol efflux, when present in HDL than in lipoprotein-deficient serum: relevance to diabetes.

Rosenblat M, Karry R, Aviram M. Rosenblat M, et al. Atherosclerosis. 2006 Jul;187(1):74-81. doi: 10.1016/j.atherosclerosis.2005.08.026. Epub 2005 Oct 17. Atherosclerosis. 2006. PMID: 16229851

10

Pomegranate byproduct administration to apolipoprotein e-deficient mice attenuates atherosclerosis development as a result of decreased macrophage oxidative stress and reduced cellular uptake of oxidized low-density lipoprotein.

Rosenblat M, Volkova N, Coleman R, Aviram M. Rosenblat M, et al. J Agric Food Chem. 2006 Mar 8;54(5):1928-35. doi: 10.1021/jf0528207. J Agric Food Chem. 2006. PMID: 16506855

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