Aviram M - Search Results

1

Paraoxonase (PON1) deficiency is associated with increased macrophage oxidative stress: studies in PON1-knockout mice.

Rozenberg O, Rosenblat M, Coleman R, Shih DM, Aviram M. Rozenberg O, et al. Among authors: aviram m. Free Radic Biol Med. 2003 Mar 15;34(6):774-84. doi: 10.1016/s0891-5849(02)01429-6. Free Radic Biol Med. 2003. PMID: 12633754

2

Oxysterol-induced activation of macrophage NADPH-oxidase enhances cell-mediated oxidation of LDL in the atherosclerotic apolipoprotein E deficient mouse: inhibitory role for vitamin E.

Rosenblat M, Aviram M. Rosenblat M, et al. Among authors: aviram m. Atherosclerosis. 2002 Jan;160(1):69-80. doi: 10.1016/s0021-9150(01)00563-9. Atherosclerosis. 2002. PMID: 11755924

3

Serum paraoxonase activity and the extent of lipid peroxidation are not affected by increased levels of human apolipoprotein A-I: studies in transgenic mice.

Rosenblat M, Grunfeld O, Hayek T, Aviram M. Rosenblat M, et al. Among authors: aviram m. Clin Chem Lab Med. 2002 Jan;40(1):9-14. doi: 10.1515/CCLM.2002.003. Clin Chem Lab Med. 2002. PMID: 11916277

4

Increased macrophage glutathione content reduces cell-mediated oxidation of LDL and atherosclerosis in apolipoprotein E-deficient mice.

Rosenblat M, Coleman R, Aviram M. Rosenblat M, et al. Among authors: aviram m. Atherosclerosis. 2002 Jul;163(1):17-28. doi: 10.1016/s0021-9150(01)00744-4. Atherosclerosis. 2002. PMID: 12048118

5

Mouse macrophage paraoxonase 2 activity is increased whereas cellular paraoxonase 3 activity is decreased under oxidative stress.

Rosenblat M, Draganov D, Watson CE, Bisgaier CL, La Du BN, Aviram M. Rosenblat M, et al. Among authors: aviram m. Arterioscler Thromb Vasc Biol. 2003 Mar 1;23(3):468-74. doi: 10.1161/01.ATV.0000059385.95664.4D. Epub 2003 Jan 30. Arterioscler Thromb Vasc Biol. 2003. PMID: 12615656

6

Human serum paraoxonase 1 decreases macrophage cholesterol biosynthesis: possible role for its phospholipase-A2-like activity and lysophosphatidylcholine formation.

Rozenberg O, Shih DM, Aviram M. Rozenberg O, et al. Among authors: aviram m. Arterioscler Thromb Vasc Biol. 2003 Mar 1;23(3):461-7. doi: 10.1161/01.ATV.0000060462.35946.B3. Epub 2003 Feb 6. Arterioscler Thromb Vasc Biol. 2003. PMID: 12615663

7

Tissue angiotensin-converting-enzyme (ACE) deficiency leads to a reduction in oxidative stress and in atherosclerosis: studies in ACE-knockout mice type 2.

Hayek T, Pavlotzky E, Hamoud S, Coleman R, Keidar S, Aviram M, Kaplan M. Hayek T, et al. Among authors: aviram m. Arterioscler Thromb Vasc Biol. 2003 Nov 1;23(11):2090-6. doi: 10.1161/01.ATV.0000098653.74209.C6. Epub 2003 Oct 2. Arterioscler Thromb Vasc Biol. 2003. PMID: 14525797

8

Decreased macrophage paraoxonase 2 expression in patients with hypercholesterolemia is the result of their increased cellular cholesterol content: effect of atorvastatin therapy.

Rosenblat M, Hayek T, Hussein K, Aviram M. Rosenblat M, et al. Among authors: aviram m. Arterioscler Thromb Vasc Biol. 2004 Jan;24(1):175-80. doi: 10.1161/01.ATV.0000104011.88939.06. Epub 2003 Oct 30. Arterioscler Thromb Vasc Biol. 2004. PMID: 14592851

9

Paraoxonases 1, 2, and 3, oxidative stress, and macrophage foam cell formation during atherosclerosis development.

Aviram M, Rosenblat M. Aviram M, et al. Free Radic Biol Med. 2004 Nov 1;37(9):1304-16. doi: 10.1016/j.freeradbiomed.2004.06.030. Free Radic Biol Med. 2004. PMID: 15454271 Review.

10

Paraoxonase 1 (PON1) enhances HDL-mediated macrophage cholesterol efflux via the ABCA1 transporter in association with increased HDL binding to the cells: a possible role for lysophosphatidylcholine.

Rosenblat M, Vaya J, Shih D, Aviram M. Rosenblat M, et al. Among authors: aviram m. Atherosclerosis. 2005 Mar;179(1):69-77. doi: 10.1016/j.atherosclerosis.2004.10.028. Epub 2004 Dec 29. Atherosclerosis. 2005. PMID: 15721011

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