Willecke K - Search Results

1

Morphology and morphometric investigation of hepatocellular preneoplastic lesions and neoplasms in connexin32-deficient mice.

Evert M, Ott T, Temme A, Willecke K, Dombrowski F. Evert M, et al. Among authors: willecke k. Carcinogenesis. 2002 May;23(5):697-703. doi: 10.1093/carcin/23.5.697. Carcinogenesis. 2002. PMID: 12016140

2

The effect of connexin32 null mutation on hepatocarcinogenesis in different mouse strains.

Moennikes O, Buchmann A, Ott T, Willecke1 K, Schwarz M. Moennikes O, et al. Carcinogenesis. 1999 Jul;20(7):1379-82. doi: 10.1093/carcin/20.7.1379. Carcinogenesis. 1999. PMID: 10383916

3

Lack of phenobarbital-mediated promotion of hepatocarcinogenesis in connexin32-null mice.

Moennikes O, Buchmann A, Romualdi A, Ott T, Werringloer J, Willecke K, Schwarz M. Moennikes O, et al. Among authors: willecke k. Cancer Res. 2000 Sep 15;60(18):5087-91. Cancer Res. 2000. PMID: 11016633

4

Ablation of gap junctional communication in hepatocytes of transgenic mice does not lead to disrupted cellular homeostasis or increased spontaneous tumourigenesis.

Ott T, Jokwitz M, Lenhard D, Romualdi A, Dombrowski F, Ittrich C, Schwarz M, Willecke K. Ott T, et al. Among authors: willecke k. Eur J Cell Biol. 2006 Aug;85(8):717-28. doi: 10.1016/j.ejcb.2006.03.004. Epub 2006 Jun 5. Eur J Cell Biol. 2006. PMID: 16740338

5

Characterization of Targeted Connexin32-Deficient Mice: A Model for the Human Charcot-Marie-Tooth (X-Type) Inherited Disease.

Willecke K, Temme A, Teubner B, Ott T. Willecke K, et al. Ann N Y Acad Sci. 1999 Oct;883(1):302-309. doi: 10.1111/j.1749-6632.1999.tb08592.x. Ann N Y Acad Sci. 1999. PMID: 29086934 No abstract available.

6

Quantitative analysis of gap-junctional intercellular communication in precision-cut mouse liver slices.

Romualdi A, Niessen H, Dombrowski F, Willecke K, Ott T. Romualdi A, et al. Among authors: willecke k. Cell Tissue Res. 2002 Mar;307(3):315-20. doi: 10.1007/s00441-002-0518-y. Epub 2002 Feb 27. Cell Tissue Res. 2002. PMID: 11904767

7

High incidence of spontaneous and chemically induced liver tumors in mice deficient for connexin32.

Temme A, Buchmann A, Gabriel HD, Nelles E, Schwarz M, Willecke K. Temme A, et al. Among authors: willecke k. Curr Biol. 1997 Sep 1;7(9):713-6. doi: 10.1016/s0960-9822(06)00302-2. Curr Biol. 1997. PMID: 9285723 Free article.

8

Downregulation of connexin32 protein and gap-junctional intercellular communication by cytokine-mediated acute-phase response in immortalized mouse hepatocytes.

Temme A, Traub O, Willecke K. Temme A, et al. Among authors: willecke k. Cell Tissue Res. 1998 Nov;294(2):345-50. doi: 10.1007/s004410051184. Cell Tissue Res. 1998. PMID: 9799450

9

Characterization of targeted connexin32-deficient mice: a model for the human Charcot-Marie-Tooth (X-type) inherited disease.

Willecke K, Temme A, Teubner B, Ott T. Willecke K, et al. Ann N Y Acad Sci. 1999 Sep 14;883:302-9. Ann N Y Acad Sci. 1999. PMID: 10586255 Review. No abstract available.

10

The extent of synchronous initiation and termination of DNA synthesis in regenerating mouse liver is dependent on connexin32 expressing gap junctions.

Temme A, Ott T, Dombrowski F, Willecke K. Temme A, et al. Among authors: willecke k. J Hepatol. 2000 Apr;32(4):627-35. doi: 10.1016/s0168-8278(00)80225-1. J Hepatol. 2000. PMID: 10782912

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