Chapman J - Search Results

1

The Ras-pathway inhibitor, S-trans-trans-farnesylthiosalicylic acid, suppresses experimental allergic encephalomyelitis.

Karussis D, Abramsky O, Grigoriadis N, Chapman J, Mizrachi-Koll R, Niv H, Kloog Y. Karussis D, et al. Among authors: chapman j. J Neuroimmunol. 2001 Nov 1;120(1-2):1-9. doi: 10.1016/s0165-5728(01)00385-x. J Neuroimmunol. 2001. PMID: 11694313

2

Lack of apolipoprotein-E exacerbates experimental allergic encephalomyelitis.

Karussis D, Michaelson DM, Grigoriadis N, Korezyn AD, Mizrachi-Koll R, Chapman S, Abramsky O, Chapman J. Karussis D, et al. Among authors: chapman s, chapman j. Mult Scler. 2003 Oct;9(5):476-80. doi: 10.1191/1352458503ms950oa. Mult Scler. 2003. PMID: 14582773

3

Increased thrombin inhibition in experimental autoimmune encephalomyelitis.

Beilin O, Karussis DM, Korczyn AD, Gurwitz D, Aronovich R, Hantai D, Grigoriadis N, Mizrachi-Kol R, Chapman J. Beilin O, et al. Among authors: chapman j. J Neurosci Res. 2005 Feb 1;79(3):351-9. doi: 10.1002/jnr.20270. J Neurosci Res. 2005. PMID: 15605378

4

Inhibition of Ras attenuates the course of experimental autoimmune neuritis.

Kafri M, Kloog Y, Korczyn AD, Ferdman-Aronovich R, Drory V, Katzav A, Wirguin I, Chapman J. Kafri M, et al. Among authors: chapman j. J Neuroimmunol. 2005 Nov;168(1-2):46-55. doi: 10.1016/j.jneuroim.2005.07.008. J Neuroimmunol. 2005. PMID: 16154640

5

The combined treatment of Copaxone and Salirasib attenuates experimental autoimmune encephalomyelitis (EAE) in mice.

Aizman E, Mor A, Chapman J, Assaf Y, Kloog Y. Aizman E, et al. Among authors: chapman j. J Neuroimmunol. 2010 Dec 15;229(1-2):192-203. doi: 10.1016/j.jneuroim.2010.08.022. Epub 2010 Sep 24. J Neuroimmunol. 2010. PMID: 20869125

6

Treatment of MRL/lpr mice, a genetic autoimmune model, with the Ras inhibitor, farnesylthiosalicylate (FTS).

Katzav A, Kloog Y, Korczyn AD, Niv H, Karussis DM, Wang N, Rabinowitz R, Blank M, Shoenfeld Y, Chapman J. Katzav A, et al. Among authors: chapman j. Clin Exp Immunol. 2001 Dec;126(3):570-7. doi: 10.1046/j.1365-2249.2001.01674.x. Clin Exp Immunol. 2001. PMID: 11737078 Free PMC article.

7

The Ras antagonist, farnesylthiosalicylic acid (FTS), decreases fibrosis and improves muscle strength in dy/dy mouse model of muscular dystrophy.

Nevo Y, Aga-Mizrachi S, Elmakayes E, Yanay N, Ettinger K, Elbaz M, Brunschwig Z, Dadush O, Elad-Sfadia G, Haklai R, Kloog Y, Chapman J, Reif S. Nevo Y, et al. Among authors: chapman j. PLoS One. 2011 Mar 22;6(3):e18049. doi: 10.1371/journal.pone.0018049. PLoS One. 2011. PMID: 21445359 Free PMC article.

8

Evidence for the genetic role of human leukocyte antigens in low frequency DRB1*1501 multiple sclerosis patients in Israel.

Karni A, Kohn Y, Safirman C, Abramsky O, Barcellos L, Oksenberg JR, Kahana E, Karussis D, Chapman J, Brautbar C. Karni A, et al. Among authors: chapman j. Mult Scler. 1999 Dec;5(6):410-5. doi: 10.1177/135245859900500i607. Mult Scler. 1999. PMID: 10618697

9

Inhibition of ras by farnesylthiosalicylate significantly reduces the levels of autoantibodies in two animal models of the antiphospholipid syndrome.

Katzav A, Kloog Y, Korczyn AD, Molina V, Blank M, Shoenfeld Y, Chapman J. Katzav A, et al. Among authors: chapman j. Immunobiology. 2003;207(1):47-50. doi: 10.1078/0171-2985-00208. Immunobiology. 2003. PMID: 12638903

10

Antiphospholipid antibodies, thrombin and LPS activate brain endothelial cells and Ras-dependent pathways through distinct mechanisms.

Aronovich R, Gurwitz D, Kloog Y, Chapman J. Aronovich R, et al. Among authors: chapman j. Immunobiology. 2005;210(10):781-8. doi: 10.1016/j.imbio.2005.10.010. Epub 2005 Oct 25. Immunobiology. 2005. PMID: 16325498

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