Walsh K - Search Results

1

Beta-amyloid peptide expression is sufficient for myotube death: implications for human inclusion body myopathy.

Querfurth HW, Suhara T, Rosen KM, McPhie DL, Fujio Y, Tejada G, Neve RL, Adelman LS, Walsh K. Querfurth HW, et al. Among authors: walsh k. Mol Cell Neurosci. 2001 May;17(5):793-810. doi: 10.1006/mcne.2001.0972. Mol Cell Neurosci. 2001. PMID: 11358479

2

Intraneuronal beta-amyloid expression downregulates the Akt survival pathway and blunts the stress response.

Magrané J, Rosen KM, Smith RC, Walsh K, Gouras GK, Querfurth HW. Magrané J, et al. Among authors: walsh k. J Neurosci. 2005 Nov 23;25(47):10960-9. doi: 10.1523/JNEUROSCI.1723-05.2005. J Neurosci. 2005. PMID: 16306409 Free PMC article.

3

Transgenic expression of beta-APP in fast-twitch skeletal muscle leads to calcium dyshomeostasis and IBM-like pathology.

Moussa CE, Fu Q, Kumar P, Shtifman A, Lopez JR, Allen PD, LaFerla F, Weinberg D, Magrane J, Aprahamian T, Walsh K, Rosen KM, Querfurth HW. Moussa CE, et al. Among authors: walsh k. FASEB J. 2006 Oct;20(12):2165-7. doi: 10.1096/fj.06-5763fje. Epub 2006 Aug 29. FASEB J. 2006. PMID: 16940437

4

Aberrant cell cycle reentry in human and experimental inclusion body myositis and polymyositis.

Kwon B, Kumar P, Lee HK, Zeng L, Walsh K, Fu Q, Barakat A, Querfurth HW. Kwon B, et al. Among authors: walsh k. Hum Mol Genet. 2014 Jul 15;23(14):3681-94. doi: 10.1093/hmg/ddu077. Epub 2014 Feb 20. Hum Mol Genet. 2014. PMID: 24556217 Free PMC article.

5

Abeta42 generation is toxic to endothelial cells and inhibits eNOS function through an Akt/GSK-3beta signaling-dependent mechanism.

Suhara T, Magrané J, Rosen K, Christensen R, Kim HS, Zheng B, McPhie DL, Walsh K, Querfurth H. Suhara T, et al. Among authors: walsh k. Neurobiol Aging. 2003 May-Jun;24(3):437-51. doi: 10.1016/s0197-4580(02)00135-5. Neurobiol Aging. 2003. PMID: 12600720

6

Heat shock protein 70 participates in the neuroprotective response to intracellularly expressed beta-amyloid in neurons.

Magrané J, Smith RC, Walsh K, Querfurth HW. Magrané J, et al. Among authors: walsh k. J Neurosci. 2004 Feb 18;24(7):1700-6. doi: 10.1523/JNEUROSCI.4330-03.2004. J Neurosci. 2004. PMID: 14973234 Free PMC article.

7

Suppression of Akt signaling induces Fas ligand expression: involvement of caspase and Jun kinase activation in Akt-mediated Fas ligand regulation.

Suhara T, Kim HS, Kirshenbaum LA, Walsh K. Suhara T, et al. Among authors: walsh k. Mol Cell Biol. 2002 Jan;22(2):680-91. doi: 10.1128/MCB.22.2.680-691.2002. Mol Cell Biol. 2002. PMID: 11756562 Free PMC article.

8

Cell cycle withdrawal promotes myogenic induction of Akt, a positive modulator of myocyte survival.

Fujio Y, Guo K, Mano T, Mitsuuchi Y, Testa JR, Walsh K. Fujio Y, et al. Among authors: walsh k. Mol Cell Biol. 1999 Jul;19(7):5073-82. doi: 10.1128/MCB.19.7.5073. Mol Cell Biol. 1999. PMID: 10373556 Free PMC article.

9

Vascular endothelial cells and smooth muscle cells differ in expression of Fas and Fas ligand and in sensitivity to Fas ligand-induced cell death: implications for vascular disease and therapy.

Sata M, Suhara T, Walsh K. Sata M, et al. Among authors: walsh k. Arterioscler Thromb Vasc Biol. 2000 Feb;20(2):309-16. doi: 10.1161/01.atv.20.2.309. Arterioscler Thromb Vasc Biol. 2000. PMID: 10669625

10

Akt mediates cytoprotection of endothelial cells by vascular endothelial growth factor in an anchorage-dependent manner.

Fujio Y, Walsh K. Fujio Y, et al. Among authors: walsh k. J Biol Chem. 1999 Jun 4;274(23):16349-54. doi: 10.1074/jbc.274.23.16349. J Biol Chem. 1999. PMID: 10347193 Free PMC article.

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