Neve RL - Search Results

1

Beta-amyloid peptide expression is sufficient for myotube death: implications for human inclusion body myopathy.

Querfurth HW, Suhara T, Rosen KM, McPhie DL, Fujio Y, Tejada G, Neve RL, Adelman LS, Walsh K. Querfurth HW, et al. Among authors: neve rl. Mol Cell Neurosci. 2001 May;17(5):793-810. doi: 10.1006/mcne.2001.0972. Mol Cell Neurosci. 2001. PMID: 11358479

2

Dysfunction of amyloid precursor protein signaling in neurons leads to DNA synthesis and apoptosis.

Neve RL, McPhie DL. Neve RL, et al. Biochim Biophys Acta. 2007 Apr;1772(4):430-7. doi: 10.1016/j.bbadis.2006.10.008. Epub 2006 Oct 18. Biochim Biophys Acta. 2007. PMID: 17113271 Free PMC article. Review.

3

Alzheimer's disease: a dysfunction of the amyloid precursor protein(1).

Neve RL, McPhie DL, Chen Y. Neve RL, et al. Brain Res. 2000 Dec 15;886(1-2):54-66. doi: 10.1016/s0006-8993(00)02869-9. Brain Res. 2000. PMID: 11119687 Review.

4

Alzheimer's disease: dysfunction of a signalling pathway mediated by the amyloid precursor protein?

Neve RL, McPhie DL, Chen Y. Neve RL, et al. Biochem Soc Symp. 2001;(67):37-50. doi: 10.1042/bss0670037. Biochem Soc Symp. 2001. PMID: 11447838 Review.

5

beta-Secretase cleavage of the amyloid precursor protein mediates neuronal apoptosis caused by familial Alzheimer's disease mutations.

McPhie DL, Golde T, Eckman CB, Yager D, Brant JB, Neve RL. McPhie DL, et al. Among authors: neve rl. Brain Res Mol Brain Res. 2001 Dec 16;97(1):103-13. doi: 10.1016/s0169-328x(01)00294-7. Brain Res Mol Brain Res. 2001. PMID: 11744168

6

Neuronal expression of beta-amyloid precursor protein Alzheimer mutations causes intracellular accumulation of a C-terminal fragment containing both the amyloid beta and cytoplasmic domains.

McPhie DL, Lee RK, Eckman CB, Olstein DH, Durham SP, Yager D, Younkin SG, Wurtman RJ, Neve RL. McPhie DL, et al. Among authors: neve rl. J Biol Chem. 1997 Oct 3;272(40):24743-6. doi: 10.1074/jbc.272.40.24743. J Biol Chem. 1997. PMID: 9312066 Free article.

7

APP-BP1 mediates APP-induced apoptosis and DNA synthesis and is increased in Alzheimer's disease brain.

Chen Y, Liu W, McPhie DL, Hassinger L, Neve RL. Chen Y, et al. Among authors: neve rl. J Cell Biol. 2003 Oct 13;163(1):27-33. doi: 10.1083/jcb.200304003. J Cell Biol. 2003. PMID: 14557245 Free PMC article.

8

The cell cycle as a therapeutic target for Alzheimer's disease.

Neve RL, McPhie DL. Neve RL, et al. Pharmacol Ther. 2006 Jul;111(1):99-113. doi: 10.1016/j.pharmthera.2005.09.005. Epub 2005 Nov 7. Pharmacol Ther. 2006. PMID: 16274748 Review.

9

Transgenic mice expressing APP-C100 in the brain.

Neve RL, Boyce FM, McPhie DL, Greenan J, Oster-Granite ML. Neve RL, et al. Neurobiol Aging. 1996 Mar-Apr;17(2):191-203. doi: 10.1016/0197-4580(95)02074-8. Neurobiol Aging. 1996. PMID: 8744400

10

Overexpression in neurons of human presenilin-1 or a presenilin-1 familial Alzheimer disease mutant does not enhance apoptosis.

Bursztajn S, DeSouza R, McPhie DL, Berman SA, Shioi J, Robakis NK, Neve RL. Bursztajn S, et al. Among authors: neve rl. J Neurosci. 1998 Dec 1;18(23):9790-9. doi: 10.1523/JNEUROSCI.18-23-09790.1998. J Neurosci. 1998. PMID: 9822738 Free PMC article.

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