David JR - Search Results

1

IL-12 gene-deficient C57BL/6 mice are susceptible to Leishmania donovani but have diminished hepatic immunopathology.

Satoskar AR, Rodig S, Telford SR 3rd, Satoskar AA, Ghosh SK, von Lichtenberg F, David JR. Satoskar AR, et al. Among authors: david jr. Eur J Immunol. 2000 Mar;30(3):834-9. doi: 10.1002/1521-4141(200003)30:3<834::AID-IMMU834>3.0.CO;2-9. Eur J Immunol. 2000. PMID: 10741399 Free article.

2

Gammadelta T cells are not essential for control of cutaneous Leishmania major infection in genetically resistant C57BL/6 mice.

Satoskar A, Okano M, David JR. Satoskar A, et al. Among authors: david jr. J Infect Dis. 1997 Dec;176(6):1649-52. doi: 10.1086/517348. J Infect Dis. 1997. PMID: 9395385

3

Enhanced Th2-like responses in IL-1 type 1 receptor-deficient mice.

Satoskar AR, Okano M, Connaughton S, Raisanen-Sokolwski A, David JR, Labow M. Satoskar AR, et al. Among authors: david jr. Eur J Immunol. 1998 Jul;28(7):2066-74. doi: 10.1002/(SICI)1521-4141(199807)28:07<2066::AID-IMMU2066>3.0.CO;2-X. Eur J Immunol. 1998. PMID: 9692874 Free article.

4

Mice with STAT6-targeted gene disruption develop a Th1 response and control cutaneous leishmaniasis.

Stamm LM, Räisänen-Sokolowski A, Okano M, Russell ME, David JR, Satoskar AR. Stamm LM, et al. Among authors: david jr. J Immunol. 1998 Dec 1;161(11):6180-8. J Immunol. 1998. PMID: 9834104

5

Mice lacking NK cells develop an efficient Th1 response and control cutaneous Leishmania major infection.

Satoskar AR, Stamm LM, Zhang X, Satoskar AA, Okano M, Terhorst C, David JR, Wang B. Satoskar AR, et al. Among authors: david jr. J Immunol. 1999 Jun 1;162(11):6747-54. J Immunol. 1999. PMID: 10352294

6

STAT-4 mediated IL-12 signaling pathway is critical for the development of protective immunity in cutaneous leishmaniasis.

Stamm LM, Satoskar AA, Ghosh SK, David JR, Satoskar AR. Stamm LM, et al. Among authors: david jr. Eur J Immunol. 1999 Aug;29(8):2524-9. doi: 10.1002/(SICI)1521-4141(199908)29:08<2524::AID-IMMU2524>3.0.CO;2-H. Eur J Immunol. 1999. PMID: 10458767 Free article.

7

Genetically resistant mice lacking IL-18 gene develop Th1 response and control cutaneous Leishmania major infection.

Monteforte GM, Takeda K, Rodriguez-Sosa M, Akira S, David JR, Satoskar AR. Monteforte GM, et al. Among authors: david jr. J Immunol. 2000 Jun 1;164(11):5890-3. doi: 10.4049/jimmunol.164.11.5890. J Immunol. 2000. PMID: 10820270

8

Migration-inhibitory factor gene-deficient mice are susceptible to cutaneous Leishmania major infection.

Satoskar AR, Bozza M, Rodriguez Sosa M, Lin G, David JR. Satoskar AR, et al. Among authors: david jr. Infect Immun. 2001 Feb;69(2):906-11. doi: 10.1128/IAI.69.2.906-911.2001. Infect Immun. 2001. PMID: 11159984 Free PMC article.

9

Cutting edge: susceptibility to the larval stage of the helminth parasite Taenia crassiceps is mediated by Th2 response induced via STAT6 signaling.

Rodriguez-Sosa M, David JR, Bojalil R, Satoskar AR, Terrazas LI. Rodriguez-Sosa M, et al. Among authors: david jr. J Immunol. 2002 Apr 1;168(7):3135-9. doi: 10.4049/jimmunol.168.7.3135. J Immunol. 2002. PMID: 11907063

10

The PACAP-type I receptor agonist maxadilan from sand fly saliva protects mice against lethal endotoxemia by a mechanism partially dependent on IL-10.

Bozza M, Soares MB, Bozza PT, Satoskar AR, Diacovo TG, Brombacher F, Titus RG, Shoemaker CB, David JR. Bozza M, et al. Among authors: david jr. Eur J Immunol. 1998 Oct;28(10):3120-7. doi: 10.1002/(SICI)1521-4141(199810)28:10<3120::AID-IMMU3120>3.0.CO;2-3. Eur J Immunol. 1998. PMID: 9808180 Free article.

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