Ott T - Search Results

1

The effect of connexin32 null mutation on hepatocarcinogenesis in different mouse strains.

Moennikes O, Buchmann A, Ott T, Willecke1 K, Schwarz M. Moennikes O, et al. Among authors: ott t. Carcinogenesis. 1999 Jul;20(7):1379-82. doi: 10.1093/carcin/20.7.1379. Carcinogenesis. 1999. PMID: 10383916

2

Morphology and morphometric investigation of hepatocellular preneoplastic lesions and neoplasms in connexin32-deficient mice.

Evert M, Ott T, Temme A, Willecke K, Dombrowski F. Evert M, et al. Among authors: ott t. Carcinogenesis. 2002 May;23(5):697-703. doi: 10.1093/carcin/23.5.697. Carcinogenesis. 2002. PMID: 12016140

3

The extent of synchronous initiation and termination of DNA synthesis in regenerating mouse liver is dependent on connexin32 expressing gap junctions.

Temme A, Ott T, Dombrowski F, Willecke K. Temme A, et al. Among authors: ott t. J Hepatol. 2000 Apr;32(4):627-35. doi: 10.1016/s0168-8278(00)80225-1. J Hepatol. 2000. PMID: 10782912

4

Lack of phenobarbital-mediated promotion of hepatocarcinogenesis in connexin32-null mice.

Moennikes O, Buchmann A, Romualdi A, Ott T, Werringloer J, Willecke K, Schwarz M. Moennikes O, et al. Among authors: ott t. Cancer Res. 2000 Sep 15;60(18):5087-91. Cancer Res. 2000. PMID: 11016633

5

Quantitative analysis of gap-junctional intercellular communication in precision-cut mouse liver slices.

Romualdi A, Niessen H, Dombrowski F, Willecke K, Ott T. Romualdi A, et al. Among authors: ott t. Cell Tissue Res. 2002 Mar;307(3):315-20. doi: 10.1007/s00441-002-0518-y. Epub 2002 Feb 27. Cell Tissue Res. 2002. PMID: 11904767

6

Effect of the tumor promoter phenobarbital on the pattern of global gene expression in liver of connexin32-wild-type and connexin32-deficient mice.

Stahl S, Ittrich C, Marx-Stoelting P, Köhle C, Ott T, Buchmann A, Schwarz M. Stahl S, et al. Among authors: ott t. Int J Cancer. 2005 Jul 20;115(6):861-9. doi: 10.1002/ijc.20815. Int J Cancer. 2005. PMID: 15751032

7

Ablation of gap junctional communication in hepatocytes of transgenic mice does not lead to disrupted cellular homeostasis or increased spontaneous tumourigenesis.

Ott T, Jokwitz M, Lenhard D, Romualdi A, Dombrowski F, Ittrich C, Schwarz M, Willecke K. Ott T, et al. Eur J Cell Biol. 2006 Aug;85(8):717-28. doi: 10.1016/j.ejcb.2006.03.004. Epub 2006 Jun 5. Eur J Cell Biol. 2006. PMID: 16740338

8

Tumor promotion in liver of mice with a conditional Cx26 knockout.

Marx-Stoelting P, Mahr J, Knorpp T, Schreiber S, Templin MF, Ott T, Buchmann A, Schwarz M. Marx-Stoelting P, et al. Among authors: ott t. Toxicol Sci. 2008 Jun;103(2):260-7. doi: 10.1093/toxsci/kfn043. Epub 2008 Feb 27. Toxicol Sci. 2008. PMID: 18308698

9

Characterization of targeted connexin32-deficient mice: a model for the human Charcot-Marie-Tooth (X-type) inherited disease.

Willecke K, Temme A, Teubner B, Ott T. Willecke K, et al. Among authors: ott t. Ann N Y Acad Sci. 1999 Sep 14;883:302-9. Ann N Y Acad Sci. 1999. PMID: 10586255 Review. No abstract available.

10

Acute-phase response and circadian expression of connexin26 are not altered in connexin32-deficient mouse liver.

Temme A, Ott T, Haberberger T, Traub O, Willecke K. Temme A, et al. Among authors: ott t. Cell Tissue Res. 2000 Apr;300(1):111-7. doi: 10.1007/s004410000177. Cell Tissue Res. 2000. PMID: 10805080

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