Hepatocyte differentiation of WIF-B cells includes a high capacity of interleukin-6-mediated induction of alpha 1-acid glycoprotein and alpha 2-macroglobulin

Biochim Biophys Acta. 1999 Jan 11;1448(3):403-8. doi: 10.1016/s0167-4889(98)00151-7.

Abstract

Responsiveness to cytokine-mediated acute inflammatory stimuli of the highly differentiated and polarized WIF-B hybrid cell line was studied by measuring the induction of alpha 1-acid glycoprotein and alpha 2-macroglobulin mRNAs after interleukin-1, interleukin-6 and tumor necrosis factor-alpha treatments in the presence of dexamethasone. Compared with their Fao parent, WIF-B cells were 10 times more responsive to 24-h interleukin-6 induction regarding alpha 2-macroglobulin induction. At variance from the response measured in Fao cells, the late effects of interleukin-6 treatment confirmed the higher sensitivity of WIF-B cells to this cytokine as a 72-h treatment as 10 times more effective than a 24-h treatment at inducting alpha 1-acid glycoprotein mRNA. These findings highlight the hepatocyte differentiation of WIF-B cells compared with other hepatoma cell lines, with respect to the regulation of acute-phase protein gene expression. They also make WIF-B cells a convenient model to study the molecular effects of interleukin-6 in terms of transduction and/or transcription, and the many cross-talks that occur during the regulation of acute-phase protein gene expression.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Differentiation / drug effects
  • Cell Line
  • Dexamethasone / pharmacology
  • Gene Expression Regulation / drug effects
  • Glucocorticoids / pharmacology
  • Interleukin-1 / pharmacology
  • Interleukin-6 / pharmacology*
  • Liver / cytology*
  • Liver / drug effects
  • Liver / metabolism*
  • Orosomucoid / biosynthesis*
  • Orosomucoid / genetics
  • RNA, Messenger / biosynthesis
  • RNA, Messenger / genetics
  • Rats
  • Tumor Necrosis Factor-alpha / pharmacology
  • alpha-Macroglobulins / biosynthesis*
  • alpha-Macroglobulins / genetics

Substances

  • Glucocorticoids
  • Interleukin-1
  • Interleukin-6
  • Orosomucoid
  • RNA, Messenger
  • Tumor Necrosis Factor-alpha
  • alpha-Macroglobulins
  • Dexamethasone