Abstract
The rate of decline of plasma HIV RNA in patients treated with anti-retroviral drugs has been postulated to reflect the half-lives of previously HIV-infected cells. Here, Zvi Grossman and colleagues argue that the observed decline is explained by the kinetics of ongoing infection cycles. Residual cell-to-cell infection that becomes increasingly difficult to block could stabilize cellular provirus reservoirs.
MeSH terms
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Anti-HIV Agents / therapeutic use*
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CD4-Positive T-Lymphocytes / virology
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Drug Therapy, Combination
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HIV / drug effects
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HIV / isolation & purification
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HIV / physiology
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HIV Infections / drug therapy*
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HIV Infections / prevention & control
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HIV Infections / virology
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Humans
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Models, Biological
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Models, Theoretical
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RNA, Viral / blood
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Viral Load
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Virus Replication / drug effects
Substances
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Anti-HIV Agents
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RNA, Viral