Abstract
This report presents a familial amyotrophic lateral sclerosis (FALS) patient with widespread vacuoles and hyaline inclusions strongly immunostained with the anti-superoxide dismutase (SOD1) antibody. The overall pathologic similarity between our non-SOD1-linked FALS patient and transgenic mice expressing a mutated human SOD1 gene suggests that common pathogenetic mechanisms other than an SOD1 mutation exist in the development of these diseases.
Publication types
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Case Reports
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Research Support, Non-U.S. Gov't
MeSH terms
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Aged
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Amyotrophic Lateral Sclerosis / genetics
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Amyotrophic Lateral Sclerosis / metabolism
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Amyotrophic Lateral Sclerosis / pathology*
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Brain / metabolism
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Brain / pathology
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Brain / ultrastructure
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Fatal Outcome
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Female
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Humans
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Immunohistochemistry
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Inclusion Bodies / pathology*
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Inclusion Bodies / ultrastructure
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Neurofilament Proteins / metabolism
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Pedigree
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Spinal Cord / pathology
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Spinal Cord / ultrastructure
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Superoxide Dismutase / genetics
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Superoxide Dismutase / metabolism
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Superoxide Dismutase-1
Substances
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Neurofilament Proteins
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SOD1 protein, human
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Sod1 protein, mouse
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Superoxide Dismutase
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Superoxide Dismutase-1