A novel complex of proteins binds the HIV-1 promoter upon virus interaction with CD4

J Biomed Sci. 1998 Jul-Aug;5(4):281-9. doi: 10.1007/BF02255860.

Abstract

HIV-1Lai13EM is a mutant isolate which is less sensitive than the parental HIV-1Lai strain to an in vitro treatment with 13B8-2 anti-CD4 monoclonal antibody (mAb) that generally inhibits transcription of HIV-1 and HIV-2. In contrast to other clade B viruses, this isolate carries a point mutation G > A at position -188 of the viral promoter. The fact that HIV-1NDK, a clade D virus insensitive to 13B8-2 mAb, also carries an A nucleotide at this position has brought our attention to the sequence surrounding position -188. Here we analyzed whether a DNA-binding molecule interacts with this region. Electrophoretic mobility shift assays performed with the -201/-175 HIV-1Lai wild-type sequence or the sequence containing a point mutation G > A at position -188 demonstrated their ability to bind a heterotrimeric complex induced in CEM cells by stimulation with heat-inactivated HIV-1.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenine
  • Antibodies, Monoclonal
  • Base Sequence
  • Binding Sites
  • CD4 Antigens / metabolism*
  • Cell Line
  • Consensus Sequence
  • Guanosine
  • HIV Long Terminal Repeat*
  • HIV-1 / genetics
  • HIV-1 / physiology*
  • Humans
  • NF-kappa B / metabolism
  • Point Mutation*
  • Promoter Regions, Genetic*
  • Regulatory Sequences, Nucleic Acid
  • Sequence Alignment
  • Transcription, Genetic
  • Virus Replication*

Substances

  • Antibodies, Monoclonal
  • CD4 Antigens
  • NF-kappa B
  • Guanosine
  • Adenine