High SDF-1 expression in HIV-1 carriers does not correlate with CD8+ T-cell-mediated suppression of viral replication

Virology. 1998 May 10;244(2):467-72. doi: 10.1006/viro.1998.9151.

Abstract

Stromal cell-derived factor 1 (SDF-1) inhibits T-cell tropic (T-tropic) HIV-1 infection in vitro. In this study, we examined the regulatory role of SDF-1 on HIV-1 replication in peripheral blood mononuclear cells (PBMC) of HIV-infected individuals. We found that the amount of SDF-1 mRNA in freshly isolated PBMC of HIV-1 carriers was higher than in healthy donors. Moreover, PBMC from some asymptomatic carriers (ACs) exhibited high levels of SDF-1 mRNA expression. The level of SDF-1 expression in PBMC did not correlate with the magnitude of CD8+ T-cell-mediated suppression of HIV-1 among ACs SDF-1 inhibited HIV-1 replication at the viral entry step, whereas a single-cycle HIV-1 infection system showed that the major part of the CD8+ T-cell-mediated suppression occurs after intracellular penetration of the virus. Our results suggest that SDF-1 acts as a suppressor of virus replication in a CD8+ T-cell-independent mechanism in HIV-infected individuals.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Base Sequence
  • CD8-Positive T-Lymphocytes / immunology*
  • Carrier State / immunology*
  • Carrier State / virology*
  • Chemokine CXCL12
  • Chemokines, CXC / genetics*
  • Chemokines, CXC / physiology
  • DNA Primers / genetics
  • Gene Expression
  • HIV Infections / immunology*
  • HIV Infections / virology*
  • HIV-1 / immunology
  • HIV-1 / physiology*
  • Humans
  • In Vitro Techniques
  • Leukocytes, Mononuclear / immunology
  • Leukocytes, Mononuclear / metabolism
  • Leukocytes, Mononuclear / virology
  • RNA, Messenger / blood
  • RNA, Messenger / genetics
  • Virus Replication / immunology

Substances

  • CXCL12 protein, human
  • Chemokine CXCL12
  • Chemokines, CXC
  • DNA Primers
  • RNA, Messenger