WAY 100,635 enhances both the 'antidepressant' actions of duloxetine and its influence on dialysate levels of serotonin in frontal cortex

Eur J Pharmacol. 1998 Jan 12;341(2-3):165-7. doi: 10.1016/s0014-2999(97)01445-3.

Abstract

The mixed serotonin and noradrenaline reuptake inhibitor, duloxetine, (5.0 mg/kg, s.c.), increased levels of serotonin (220%), dopamine (180%) and noradrenaline (470%) in individual dialysates of frontal cortex of freely moving rats. Its influence on serotonin, but not dopamine or noradrenaline, levels was enhanced by the 5-HT1A receptor antagonist, WAY 100,635 (N-[2-[4-(2-methoxyphenyl)-1-piperazinyl]ethyl]-N-(2-pyridinyl) cyclo-hexanecarboxamide 3HCl) (0.16 mg kg(-1), s.c). In the forced swimming test, although duloxetine was inactive alone, it dose dependently reduced immobility in the presence of WAY 100,635. Thus, blockade of 5-HT1A (auto)receptors selectively facilitates the influence of duloxetine on serotonin levels in the frontal cortex in rats and, in the forced swimming model, enhances its 'antidepressant' properties in parallel.

MeSH terms

  • Animals
  • Antidepressive Agents / pharmacology*
  • Cerebral Cortex / drug effects*
  • Cerebral Cortex / metabolism
  • Drug Synergism
  • Duloxetine Hydrochloride
  • Frontal Lobe / drug effects
  • Frontal Lobe / metabolism
  • Male
  • Piperazines / pharmacology*
  • Pyridines / pharmacology*
  • Rats
  • Rats, Wistar
  • Serotonin / metabolism*
  • Serotonin Antagonists / pharmacology*
  • Thiophenes / pharmacology*

Substances

  • Antidepressive Agents
  • Piperazines
  • Pyridines
  • Serotonin Antagonists
  • Thiophenes
  • Serotonin
  • N-(2-(4-(2-methoxyphenyl)-1-piperazinyl)ethyl)-N-(2-pyridinyl)cyclohexanecarboxamide
  • Duloxetine Hydrochloride