Abstract
Molecular genetic investigation of a female infant with Beckwith-Wiedemann syndrome (BWS) showed loss of IGF2 imprinting but no evidence of uniparental disomy. In addition, a deletion of chromosome 18q22.1 was identified in this infant without clinical features of 18q-syndrome (microcephaly, short stature, hypotonia). The association of a chromosome 18 deletion and BWS may be coincidental or may indicate the location of a trans activating regulator element for maintenance of IGF2 imprinting.
MeSH terms
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Beckwith-Wiedemann Syndrome / genetics*
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Beckwith-Wiedemann Syndrome / physiopathology
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Chromosome Deletion*
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Chromosomes, Human, Pair 18 / genetics*
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Ear, External / abnormalities
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Facial Neoplasms / genetics
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Female
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Genomic Imprinting
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Hemangioma, Capillary / genetics
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Hernia, Umbilical / genetics
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Humans
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Infant, Newborn
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Infant, Premature
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Insulin-Like Growth Factor II / genetics*
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Insulin-Like Growth Factor II / physiology
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Karyotyping
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Macroglossia / genetics
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Male
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Polymerase Chain Reaction
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Polymorphism, Genetic
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Pregnancy
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Pregnancy Complications
Substances
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Insulin-Like Growth Factor II