Mapping the binding site for matrix metalloproteinase on the N-terminal domain of the tissue inhibitor of metalloproteinases-2 by NMR chemical shift perturbation

Biochemistry. 1997 Nov 11;36(45):13882-9. doi: 10.1021/bi9712091.

Abstract

Changes in the NMR chemical shift of backbone amide nuclei (1H and 15N) have been used to map the matrix metalloproteinase (MMP) binding site on the N-terminal domain of the tissue inhibitor of metalloproteinase-2 (N-TIMP-2). Amide chemical shift changes were measured on formation of a stable complex with the catalytic domain of stromelysin-1 (N-MMP-3). Residues with significantly shifted amide signals mapped specifically to a broad site covering one face of the molecule. This site (the MMP binding site) consists primarily of residues 1-11, 27-41, 68-73, 87-90, and 97-104. The site overlaps with the OB-fold binding site seen in other proteins that share the same five-stranded beta-barrel topology. Sequence conservation data and recent site-directed mutagenesis studies are discussed in relation to the MMP binding site identified in this work.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Binding Sites / genetics
  • Carbon Isotopes
  • Conserved Sequence / genetics
  • Humans
  • Hydrogen
  • Magnetic Resonance Spectroscopy
  • Matrix Metalloproteinase 3 / chemistry
  • Matrix Metalloproteinase 3 / metabolism
  • Metalloendopeptidases / chemistry*
  • Metalloendopeptidases / genetics
  • Metalloendopeptidases / metabolism*
  • Molecular Sequence Data
  • Mutagenesis, Site-Directed
  • Nitrogen Isotopes
  • Peptide Mapping / methods*
  • Tissue Inhibitor of Metalloproteinase-2 / chemistry*
  • Tissue Inhibitor of Metalloproteinase-2 / genetics
  • Tissue Inhibitor of Metalloproteinase-2 / metabolism*

Substances

  • Carbon Isotopes
  • Nitrogen Isotopes
  • Tissue Inhibitor of Metalloproteinase-2
  • Hydrogen
  • Metalloendopeptidases
  • Matrix Metalloproteinase 3