Human heart failure: determinants of ventricular dysfunction

Adv Exp Med Biol. 1997:430:97-108. doi: 10.1007/978-1-4615-5959-7_9.

Abstract

Thin muscle strips were obtained from non-failing (NF) and failing (dilated cardiomyopathy (DCM)) hearts, using a new harvesting and dissection technique. The strips were used to carry out a myothermal and mechanical analysis so that contractile and excitation coupling phenomena in the NF and failing (DCM-F) preparations can be compared. Peak isometric force and rate of relaxation in DCM-F were reduced 46% (p < 0.02) while time to peak tension was increased 14% (p < 0.03). Initial, tension dependent, tension independent and the rate of tension independent heat liberation were reduced 62-70% in DCM-F (p < 0.03). The crossbridge force-time integral (FTIXBr) was calculated from these measurements and was shown to increase 40% while the amount and rate of calcium cycled per beat was reduced 70%. As a result of these changes in the contractile and excitation-contraction coupling systems in DCM-F, the force-frequency relationship was significantly blunted while the power output was markedly reduced. These fundamental alterations account for the substantial ventricular dysfunction found in the dilated cardiomyopathic failing heart.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Biomechanical Phenomena
  • Calcium / metabolism
  • Cardiomyopathy, Dilated / physiopathology*
  • Heart Ventricles / physiopathology
  • Humans
  • In Vitro Techniques
  • Isometric Contraction
  • Kinetics
  • Myocardial Contraction*
  • Sarcomeres / physiology
  • Thermodynamics
  • Ventricular Dysfunction / physiopathology*

Substances

  • Calcium