MHC-dependent cytolysis of autologous tumor cells by lymphocytes infiltrating urothelial carcinomas

Int J Cancer. 1997 May 16;71(4):585-94. doi: 10.1002/(sici)1097-0215(19970516)71:4<585::aid-ijc13>3.0.co;2-b.

Abstract

Tumor-infiltrating lymphocytes (TIL) were grown from 23 urothelial carcinomas. Phenotyping analysis showed that the TIL cultures were mainly CD3+. Although CD4+ and CD8+ T-cell sub-sets were grown in culture, CD4+ T-cell sub-sets predominated over CD8+ T cells. Immunohistochemical studies performed on 5 tumor specimens confirmed this observation, and indicated that CD4+ T cells surrounded the tumor islets, whereas CD8+ T lymphocytes were localized among the tumor cells. Five short-term carcinoma cell lines established from these urothelial tumors were used as target cells in cytolysis assays in order to investigate the functional anti-tumor activity of autologous TIL. TIL from 4/5 tumors were lytic and 3 TIL lines displayed MHC-class-I-dependent cytotoxicity directed against autologous tumor cells. CD4+ T-cell-depletion experiments performed on TIL line 07 confirmed that CD8+ MHC-class-I-dependent CTL were the predominant effectors. Finally, experiments performed on 6 allogeneic urothelial-cancer cell lines matched for HLA-class-I molecules showed that TIL07 exhibited selective lytic activity toward tumor 07. These data indicate that CD8+ MHC-class-I-dependent CTL present in urothelial carcinomas are functional and may participate in the anti-tumor immune response.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • CD3 Complex
  • CD4-Positive T-Lymphocytes / immunology
  • CD8-Positive T-Lymphocytes / immunology
  • Carcinoma, Transitional Cell / immunology
  • Carcinoma, Transitional Cell / pathology
  • Cytotoxicity, Immunologic
  • HLA Antigens / immunology*
  • Humans
  • Lymphocyte Subsets / immunology*
  • Lymphocytes, Tumor-Infiltrating / immunology*
  • Tumor Cells, Cultured
  • Urinary Bladder Neoplasms / immunology
  • Urinary Bladder Neoplasms / pathology
  • Urologic Neoplasms / immunology*
  • Urologic Neoplasms / pathology

Substances

  • CD3 Complex
  • HLA Antigens