Topical interleukin 1 receptor antagonist promotes corneal transplant survival

Transplantation. 1997 May 27;63(10):1501-7. doi: 10.1097/00007890-199705270-00022.

Abstract

Background: Interleukin (IL)-1 is a potent proinflammatory cytokine that plays a critical role in initiating and maintaining immunogenic inflammation. We performed a series of experiments to determine whether the topical application of IL-1 receptor antagonist (IL-1ra) can prolong corneal transplant survival in the murine model of orthotopic allotransplantation.

Methods: For all experiments, C57BL/6 corneas were transplanted into BALB/c (major histocompatibility and minor histocompatibility-disparate) eyes. "High-risk" transplants were transplants that had been sutured into BALB/c recipient beds with corneal neovascularization induced by placement of three interrupted sutures in the host cornea 2 weeks earlier. Both risk groups were divided in a masked fashion into treatment subgroups that received either 20 mg/ml of IL-1ra mixed in 0.2% sodium hyaluronate vehicle (n=28) or placebo alone (n=25). All transplants were evaluated for 8 weeks after surgery for signs of rejection. At the end of follow-up, corneal specimens were processed for enumeration of Langerhans cells and histopathological evaluation.

Results: Survival rates of both normal-risk and high-risk transplants increased significantly among the IL-1ra-treated animals compared with untreated controls by both stratified Mantel-Haenszel (P=0.02) and Kaplan-Meier survival (P=0.03) analyses. Furthermore, both normal- and high-risk IL-1ra-treated grafts had significantly less inflammation and Langerhans cells infiltration compared with untreated controls.

Conclusions: Topical treatment with IL-1ra has a significantly positive effect in promoting corneal allograft survival.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Administration, Topical
  • Animals
  • Cornea / blood supply
  • Cornea / cytology
  • Corneal Transplantation / immunology*
  • Graft Survival / drug effects
  • Interleukin 1 Receptor Antagonist Protein
  • Keratitis / drug therapy
  • Keratitis / pathology
  • Langerhans Cells / cytology
  • Mice
  • Mice, Inbred BALB C
  • Neovascularization, Physiologic
  • Receptors, Interleukin-1 / antagonists & inhibitors
  • Recombinant Proteins / pharmacology
  • Sialoglycoproteins / pharmacology*

Substances

  • Il1rn protein, mouse
  • Interleukin 1 Receptor Antagonist Protein
  • Receptors, Interleukin-1
  • Recombinant Proteins
  • Sialoglycoproteins