Cortical neurones exhibiting kainate-activated Co2+ uptake are selectively vulnerable to AMPA/kainate receptor-mediated toxicity

Neurobiol Dis. 1994 Dec;1(3):101-10. doi: 10.1006/nbdi.1994.0013.

Abstract

Kainate-activated Co2+ uptake, a histochemical method that identifies cells bearing Ca2+-permeable AMPA/kainate receptors, labels approximately 15% of murine cortical neurones in cell culture. While exposure times exceeding several hours were needed for AMPA or kainate to destroy most cultured cortical neurones, the subpopulation exhibiting kainate-activated Co2+ uptake was selectively destroyed after AMPA or kainate exposures of only 10-60 min. No selective loss was seen after exposure to NMDA. Kainate toxicity on Co2+ uptake-positive neurones was dependent on extracellular Ca2+ concentration, and associated with an increase in intracellular free Ca2+ even in the absence of extracellular Na+. These results suggest that a distinct subpopulation of cortical neurones expresses AMPA/kainate receptors linked to Ca2+-permeable channels, and that this characteristic conveys enhanced vulnerability to kainate-induced, Ca2+-mediated, damage.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Calcium / metabolism
  • Cells, Cultured
  • Cerebral Cortex / drug effects*
  • Cerebral Cortex / metabolism
  • Cobalt / pharmacokinetics*
  • Cobalt / toxicity
  • Excitatory Amino Acid Agonists / toxicity*
  • Kainic Acid / toxicity*
  • Mice
  • N-Methylaspartate / toxicity
  • Receptors, AMPA / physiology*
  • Receptors, Kainic Acid / physiology*

Substances

  • Excitatory Amino Acid Agonists
  • Receptors, AMPA
  • Receptors, Kainic Acid
  • Cobalt
  • N-Methylaspartate
  • Kainic Acid
  • Calcium