Regulation of expression of leptin mRNA and secretion of leptin by thyroid hormone in 3T3-L1 adipocytes

Biochem Biophys Res Commun. 1997 Mar 27;232(3):822-6. doi: 10.1006/bbrc.1997.6378.

Abstract

Leptin, the obese gene product, is secreted exclusively by adipocytes and regulates energy balance. We examined the effects of thyroid hormones on the regulation of leptin in 3T3-L1 adipocytes. Fully differentiated adipocytes were incubated with thyroid hormones, and the expression of leptin mRNA was measured by quantitative reverse transcription-polymerase chain reaction. After a 24-h incubation, triiodothyronine (T3) at 10-1000 nmol/l significantly increased the expression of leptin mRNA (237-337%). These stimulatory effects were not observed in preadipocytes. By contrast, thyroxine (T4) at 1-1000 nmol/l did not affect leptin mRNA expression in adipocytes. We also measured the levels of secreted leptin in conditioned media using radioimmunoassay. T3 at 10 and 1000 nmol/l significantly increased the levels of secreted leptin (132% and 126%, respectively) after a 24-h incubation. Our present data suggest that thyroid hormone is a novel regulator of leptin mRNA expression and protein secretion in 3T3-L1 adipocytes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 3T3 Cells
  • Adipose Tissue / drug effects*
  • Adipose Tissue / metabolism*
  • Animals
  • Base Sequence
  • DNA Primers / genetics
  • Dose-Response Relationship, Drug
  • Gene Expression Regulation / drug effects
  • Kinetics
  • Leptin
  • Mice
  • Polymerase Chain Reaction
  • Proteins / genetics*
  • Proteins / metabolism*
  • RNA, Messenger / genetics*
  • RNA, Messenger / metabolism*
  • Thyroid Hormones / pharmacology*
  • Thyroxine / pharmacology
  • Triiodothyronine / administration & dosage
  • Triiodothyronine / pharmacology

Substances

  • DNA Primers
  • Leptin
  • Proteins
  • RNA, Messenger
  • Thyroid Hormones
  • Triiodothyronine
  • Thyroxine