Downregulation of the PTH/PTHrP receptor by vitamin D3 in the osteoblast-like ROS 17/2.8 cells

Am J Physiol. 1996 Apr;270(4 Pt 1):E654-60. doi: 10.1152/ajpendo.1996.270.4.E654.

Abstract

Effects of 1,25-dihydroxyvitamin D3 [1,25(OH)2D3] on the expression of the parathyroid hormone (PTH)/PTH-related peptide (rP) receptor protein and mRNA in ROS 17/2.8 cells were studied. Treatment of ROS 17/2.8 cells with 1,25(OH)2D3 caused time- and dose-dependent suppression of PTH/PTHrP receptor number and immunoreactivity. The effects required more than 24 h incubation with 1,25(OH)2D3 and were maximal by 72 h. The cells did not recover their PTH/PTHrP receptors even after 4 days of treatment with control medium. Treatment with low concentrations of 1,25(OH)2D3 (0.1 M) dramatically decreased the PTH/PTHrP receptor mRNA levels, which were maximal after 24 h of incubation. The half-life of the PTH/PTHrP receptor transcript, 6-8 h, was similar in control and 1,25(OH)2D3-treated cells, suggesting that 1,25(OH)2D3 acts in controlling transcription of the PTH/PTHrP receptor gene but does not change the degradation rate of the PTH/PTHrP receptor transcripts. These data indicate that 1,25(OH)2D3 has a potent inhibitory effect on the expression of the PTH/PTHrP receptor protein and mRNA in ROS 17/2.8 cells.

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Antibodies / immunology
  • Calcitriol / pharmacology*
  • Cell Membrane / metabolism
  • Down-Regulation*
  • Molecular Sequence Data
  • Osteoblasts / drug effects*
  • Osteoblasts / metabolism*
  • Peptide Fragments / genetics
  • Peptide Fragments / immunology
  • RNA, Messenger / metabolism
  • Rats
  • Receptor, Parathyroid Hormone, Type 1
  • Receptors, Parathyroid Hormone / drug effects
  • Receptors, Parathyroid Hormone / genetics
  • Receptors, Parathyroid Hormone / metabolism*
  • Tumor Cells, Cultured

Substances

  • Antibodies
  • Peptide Fragments
  • RNA, Messenger
  • Receptor, Parathyroid Hormone, Type 1
  • Receptors, Parathyroid Hormone
  • Calcitriol