African swine fever virus gene A179L, a viral homologue of bcl-2, protects cells from programmed cell death

Virology. 1996 Nov 1;225(1):227-30. doi: 10.1006/viro.1996.0592.

Abstract

The African swine fever virus (ASFV) open reading frame A179L, which is similar to the human proto-oncogene bcl-2, has been cloned and expressed in vaccinia virus under control of the pEIL synthetic early/late promoter. The A179L gene product prevented cell death in HeLa and BSC-40 cells doubly infected with another recombinant vaccinia virus expressing the interferon-induced double-stranded RNA-activated protein kinase (p68 kinase), which activates a rapid cell death characteristic of apoptosis. This finding suggests that the A179L gene has a function similar to that of bcl-2 in preventing apoptosis and may play an important role during productive ASFV infection.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • African Swine Fever Virus / genetics*
  • Apoptosis / physiology*
  • Cells, Cultured
  • Cloning, Molecular
  • DNA Fragmentation
  • Gene Expression
  • Genes, Viral / genetics
  • Genes, Viral / physiology*
  • Genes, bcl-2 / genetics*
  • HeLa Cells
  • Humans
  • Open Reading Frames / genetics
  • Protein Serine-Threonine Kinases / genetics
  • Protein Serine-Threonine Kinases / physiology
  • Proto-Oncogene Mas
  • Sequence Analysis, DNA
  • Sequence Homology, Nucleic Acid
  • Vaccinia virus / genetics
  • eIF-2 Kinase

Substances

  • MAS1 protein, human
  • Proto-Oncogene Mas
  • Protein Serine-Threonine Kinases
  • eIF-2 Kinase