Expression of CD44 splice variants in metastatic and non-metastatic mouse tumour cell lines

Immunol Lett. 1996 Sep;52(2-3):81-7. doi: 10.1016/0165-2478(96)02588-6.

Abstract

Different splice variants of the CD44 cell-surface molecule have been linked to metastasis formation in several animal and human cancers. We have used metastatic CSML-100 and non-metastatic CSML-0 mouse adenocarcinoma cell lines to determine whether variant CD44 molecules could be implicated in the different behaviour of these cells. Two CD44 splice variants containing exons v7-v10 and v8-v10 were detected in the non-metastatic CSML-0. Two other mouse cell lines, the normal mammary gland NMuMG and the mammary pre-neoplastic CL-S1 were also found to express these exons. A short (hematopoietic) CD44 isoform was expressed in the metastatic CSML-100 and three other mouse mammary tumour cell lines. Overexpression of v7-v10 and v8-v10 CD44 variants in CSML 100 cells did not decrease their metastatic potential.

MeSH terms

  • Alternative Splicing / genetics
  • Amino Acid Sequence
  • Animals
  • Antigenic Variation*
  • Base Sequence
  • DNA Primers / genetics
  • DNA, Neoplasm / genetics
  • Female
  • Gene Expression
  • Humans
  • Hyaluronan Receptors / genetics*
  • Mammary Neoplasms, Experimental / genetics
  • Mammary Neoplasms, Experimental / immunology
  • Mammary Neoplasms, Experimental / secondary
  • Mice
  • Molecular Sequence Data
  • Neoplasm Metastasis / genetics
  • Neoplasm Metastasis / immunology*
  • Tumor Cells, Cultured

Substances

  • DNA Primers
  • DNA, Neoplasm
  • Hyaluronan Receptors