Ca2+ depletion facilitates taurine release in cultured rat astrocytes

Jpn J Pharmacol. 1996 Sep;72(1):75-8. doi: 10.1254/jjp.72.75.

Abstract

Removal of external Ca2+ facilitated endogenous taurine release in cultured rat astrocytes. The stimulated release was not affected by furosemide, sucrose, tetrodotoxin and 3,4-dichlorobenzamil, but partially inhibited by nifedipine. Omission of external Na+ increased basal taurine release, and the effects of Na+ removal and Ca2+ depletion on the release were additive. The Na(+)-free condition did not affect Ca2+ paradox-induced cell death in astrocytes. These findings suggest that Ca2+ depletion facilitates taurine release in a mechanism independent of volume and the Na+ gradient and that the release is not involved in Ca2+ paradox-induced delayed cell toxicity in astrocytes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amiloride / analogs & derivatives
  • Amiloride / pharmacology
  • Animals
  • Animals, Newborn
  • Astrocytes / drug effects*
  • Astrocytes / metabolism
  • Calcium / pharmacology*
  • Calcium Channel Blockers / pharmacology*
  • Carrier Proteins / metabolism
  • Cells, Cultured / drug effects
  • Culture Media / chemistry
  • Nifedipine / pharmacology*
  • Rats
  • Rats, Sprague-Dawley
  • Sodium / metabolism
  • Taurine / antagonists & inhibitors
  • Taurine / metabolism*

Substances

  • Calcium Channel Blockers
  • Carrier Proteins
  • Culture Media
  • 3',4'-dichlorobenzamil
  • Taurine
  • Amiloride
  • Sodium
  • Nifedipine
  • Calcium