The cytocidal activity of OK-432-activated mononuclear cells against human glioma cells is partly mediated through the Fas ligand/Fas system

Jpn J Cancer Res. 1996 Sep;87(9):972-6. doi: 10.1111/j.1349-7006.1996.tb02128.x.

Abstract

We have been applying an adoptive immunotherapy protocol to patients with malignant brain tumors using OK-432-activated peripheral blood mononuclear cells (OK-MCs). In order to elucidate the mechanism of OK-MCs' cytotoxicity, we examined the expression of Fas ligand mRNA in OK-MCs and the cytocidal activity of these cells against a human glioma cell line, T98G which expresses a high level of Fas. The expression of Fas ligand mRNA was low in non-treated peripheral blood mononuclear cells and was elevated by treatment with OK-432, irrespective of the dose employed. Apoptosis of T98G cells induced by OK-MCs was unequivocally inhibited by the pretreatment of T98 G cells with ZB4 monoclonal antibody, which binds to Fas and blocks the binding of Fas ligand to Fas. These data indicate that the cytotoxic activity of OK-MCs via apoptosis seems to be at least partly mediated by the Fas ligand/Fas system. Adoptive immunotherapy using the Fas ligand/Fas system could be a new treatment modality for human malignant brain tumors.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adjuvants, Immunologic / pharmacology*
  • Adult
  • Antibodies, Monoclonal / pharmacology
  • Apoptosis
  • Brain Neoplasms / therapy*
  • DNA Damage
  • Fas Ligand Protein
  • Glioma / therapy*
  • Humans
  • Immunotherapy, Adoptive*
  • Leukocytes, Mononuclear / drug effects*
  • Leukocytes, Mononuclear / immunology*
  • Membrane Glycoproteins / metabolism
  • Membrane Glycoproteins / physiology*
  • Picibanil / pharmacology*
  • RNA, Messenger / metabolism
  • Tumor Cells, Cultured
  • fas Receptor / metabolism
  • fas Receptor / physiology*

Substances

  • Adjuvants, Immunologic
  • Antibodies, Monoclonal
  • FASLG protein, human
  • Fas Ligand Protein
  • Membrane Glycoproteins
  • RNA, Messenger
  • fas Receptor
  • Picibanil