Cystatin C and apolipoprotein E immunoreactivities in CA1 neurons in ischemic gerbil hippocampus

Brain Res. 1996 Feb 19;709(2):155-62. doi: 10.1016/0006-8993(95)01232-x.

Abstract

The distribution patterns of cystatin C and apolipoprotein E (apo E) were studied immunocytochemically in the gerbil hippocampus before and after 5 min ischemia. In the controls, cystatin C was distributed mainly in astrocytes. In addition, a large number of dots positive for cystatin C were observed around the outlines of neuronal perikarya in the CA1 subfields. One day after ischemia, cystatin C-positive stainings outlining neuronal cell bodies disappeared. On the fourth day, intense stainings for cystatin C appeared in atrophied pyramidal neurons and these stainings in neurons disappeared by the 14th day. A remarkable increase in the number of cystatin C-positive astrocytes occurred on the fourth day and thereafter these spread over the whole of the CA1 subfield. Apo E was also distributed in astrocytes in the control specimens. From the fourth day, extra- and/or intracellular distribution of apo E-immunoreactivities was noted in the stratum pyramidale. Apo E-positive astrocytes disappeared transiently on the fourth day and then reappeared and increased remarkably by the 14th day. These findings indicate that cystatin C and apo E are involved in the degeneration process of brain neuronal cells.

MeSH terms

  • Animals
  • Apolipoproteins E / metabolism*
  • Brain Ischemia / metabolism*
  • Brain Ischemia / pathology
  • Cystatin C
  • Cystatins / metabolism*
  • Cysteine Proteinase Inhibitors / metabolism*
  • Gerbillinae
  • Glial Fibrillary Acidic Protein / metabolism
  • Hippocampus / metabolism*
  • Immunohistochemistry
  • Male
  • Neurons / metabolism*
  • Pyramidal Cells / pathology
  • Time Factors

Substances

  • Apolipoproteins E
  • Cystatin C
  • Cystatins
  • Cysteine Proteinase Inhibitors
  • Glial Fibrillary Acidic Protein