High expression of Bcl-x protein in the developing human cerebellar cortex

J Neurosci Res. 1996 Jan 15;43(2):175-82. doi: 10.1002/(SICI)1097-4547(19960115)43:2<175::AID-JNR5>3.0.CO;2-D.

Abstract

The gene bcl-x, which is related to a bcl-2, regulates programmed cell death. bcl-x may function in the development of the nervous system. We raised a polyclonal antibody against human Bcl-x protein, and investigated its distribution in the developing human cerebellum. Western blotting revealed that Bcl-x expression in the cerebellum is higher in the fetal, than in the postnatal period. Immunohistochemical studies of fetal brains localized intense Bcl-x immunoreactivity in the granule cell processes at 13-22 gestational weeks and in the Purkinje cell bodies at 24-38 weeks. The immunoreactivity decreased after birth, but was retained in the Purkinje cells at a low level until adulthood. These results suggested that Bcl-x expression in the cerebellum is developmentally regulated and involved specifically in the development of neuronal subpopulations.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Amino Acid Sequence
  • Antibodies, Monoclonal
  • Apoptosis / physiology
  • Blotting, Western
  • Cerebellar Cortex / growth & development
  • Cerebellar Cortex / metabolism*
  • Enzyme-Linked Immunosorbent Assay
  • Female
  • Humans
  • Immunoenzyme Techniques
  • Immunohistochemistry
  • Infant
  • Infant, Newborn
  • Molecular Sequence Data
  • Pregnancy
  • Proto-Oncogene Proteins / biosynthesis*
  • Proto-Oncogene Proteins c-bcl-2*
  • Thymus Gland / metabolism
  • bcl-X Protein

Substances

  • Antibodies, Monoclonal
  • BCL2L1 protein, human
  • Proto-Oncogene Proteins
  • Proto-Oncogene Proteins c-bcl-2
  • bcl-X Protein