Expression of granzyme A and perforin in mouse heart transplants immunosuppressed with donor-specific transfusion and anti-CD4 monoclonal antibody

Transplantation. 1996 Feb 27;61(4):625-9. doi: 10.1097/00007890-199602270-00018.

Abstract

Granzyme A and perforin are produced by activated cytotoxic T lymphocytes and their expression correlates with the appearance of cytotoxicity. Using in situ hybridization and immunohistochemistry, we examined the phenotype of cellular infiltration and the appearance of granzyme A+ and perforin+ cells in a mouse cardiac transplant model where the recipients were pretreated with donor-specific transfusion, anti-CD4 monoclonal antibody, or both. While the profiles of cellular infiltration failed to correlate with graft survival, tolerized grafts, as compared with untreated allografts, showed a decreased frequency of granzyme A and perforin expression. These functional markers of cytotoxic T lymphocytes can differentiate between rejecting and indefinitely surviving grafts and may be of value in dissecting the immunological events involved in tolerance induction.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acute Disease
  • Animals
  • Antibodies, Monoclonal / pharmacology*
  • Blood Transfusion*
  • CD4 Antigens / immunology*
  • Graft Rejection / immunology
  • Graft Rejection / prevention & control
  • Granzymes
  • Heart Transplantation / immunology*
  • Immune Tolerance / immunology
  • In Situ Hybridization
  • Membrane Glycoproteins / biosynthesis*
  • Membrane Glycoproteins / immunology
  • Mice
  • Mice, Inbred C3H
  • Mice, Inbred C57BL
  • Perforin
  • Pore Forming Cytotoxic Proteins
  • Rats
  • Serine Endopeptidases / biosynthesis*
  • Serine Endopeptidases / immunology
  • T-Lymphocytes, Cytotoxic / immunology

Substances

  • Antibodies, Monoclonal
  • CD4 Antigens
  • Membrane Glycoproteins
  • Pore Forming Cytotoxic Proteins
  • Perforin
  • Granzymes
  • Serine Endopeptidases