Antibacterial action of the urease inhibitor acetohydroxamic acid on Helicobacter pylori

J Clin Pathol. 1993 Apr;46(4):372-3. doi: 10.1136/jcp.46.4.372.

Abstract

The urease inhibitor acetohydroxamic acid (AHA) was assessed for its bacteriostatic and bactericidal effects on Helicobacter pylori. For eight isolates of H pylori, the minimum inhibitory concentration (MIC) was either 200 mg/l or 400 mg/l. Interactions between AHA and antimicrobial drugs used to treat H pylori were also determined. For most isolates AHA reduced the MIC for colloidal bismuth subcitrate (CBS), tetracycline, metronidazole, and amoxicillin. In a few isolates, however, AHA increased the minimum bactericidal concentration (MBC) for these antimicrobial treatments. In vitro AHA is active against H pylori and it interacts with other agents directed against H pylori.

MeSH terms

  • Amoxicillin / pharmacology
  • Anti-Ulcer Agents / pharmacology
  • Drug Interactions
  • Helicobacter pylori / drug effects*
  • Humans
  • Hydroxamic Acids / pharmacology*
  • In Vitro Techniques
  • Metronidazole / pharmacology
  • Microbial Sensitivity Tests
  • Organometallic Compounds / pharmacology
  • Tetracycline / pharmacology

Substances

  • Anti-Ulcer Agents
  • Hydroxamic Acids
  • Organometallic Compounds
  • Metronidazole
  • acetohydroxamic acid
  • Amoxicillin
  • Tetracycline
  • bismuth tripotassium dicitrate