Abstract
Each peptide CO-NH function in the biologically important C-terminal 8-13 sequence of neurotensin was replaced by the reduced CH2-NH isostere using the rapid in situ solid phase procedure developed by Sasaki & Coy. In general this modification resulted in a drop in receptor affinity except for the [Arg psi(CH2NH) Arg]-NT8-13 analogue (PIC50 9.23 vs. NT8-13 pIC50 8.03). This analogue also showed enhanced enzymatic stability, but acted as a full agonist as shown by the observation of relaxations of guinea-pig colon ascendens.
MeSH terms
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Amino Acid Sequence
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Animals
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Colon / drug effects
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Guinea Pigs
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In Vitro Techniques
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Molecular Sequence Data
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Muscle Relaxation / drug effects
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Neurotensin / analogs & derivatives*
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Neurotensin / chemical synthesis
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Neurotensin / pharmacology
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Oligopeptides / chemical synthesis
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Oligopeptides / chemistry
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Oligopeptides / pharmacology
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Oxidation-Reduction
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Peptide Fragments / chemical synthesis
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Peptide Fragments / chemistry
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Peptide Fragments / pharmacology
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Prosencephalon / metabolism
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Rats
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Structure-Activity Relationship
Substances
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Oligopeptides
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Peptide Fragments
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Neurotensin