Interleukin 1 receptor on fibroblasts from systemic sclerosis patients induces excessive functional responses to interleukin 1 beta

Biochem Biophys Res Commun. 1993 Jan 15;190(1):154-61. doi: 10.1006/bbrc.1993.1024.

Abstract

To determine if SSc fibroblasts are more sensitive to exogenous IL-1 than are normal fibroblasts, we studied the inductions of IL-6, prostaglandin E2 (PGE2), IL-1 beta mRNA production by IL-1-stimulated SSc and normal fibroblasts. The minimal IL-1 beta concentrations for stimulation of IL-6 and PGE2 production by SSc fibroblasts were 10-fold and 100-fold lower, respectively, than those for normal fibroblasts. The minimal IL-1 beta concentration for stimulation of IL-1 beta mRNA expression by SSc fibroblasts was also 10-fold lower than that for normal fibroblasts. These results suggest that the IL-1 signal transduction through IL-1R could be induced excessively in SSc fibroblasts. It is suggested that the IL-1 signal transduction plays an important role in the abnormal cytokine networks observed in the fibrosis of SSc.

MeSH terms

  • Base Sequence
  • Cells, Cultured
  • Dinoprostone / metabolism*
  • Enzyme-Linked Immunosorbent Assay
  • Fibroblasts / drug effects
  • Fibroblasts / immunology
  • Humans
  • Interleukin-1 / pharmacology*
  • Interleukin-6 / biosynthesis*
  • Interleukin-6 / genetics
  • Kinetics
  • Molecular Sequence Data
  • Oligodeoxyribonucleotides
  • Oligonucleotides, Antisense
  • Polymerase Chain Reaction / methods
  • RNA, Messenger / biosynthesis
  • RNA, Messenger / isolation & purification
  • Receptors, Interleukin-1 / drug effects
  • Receptors, Interleukin-1 / physiology*
  • Reference Values
  • Scleroderma, Systemic / immunology*
  • Skin / immunology*

Substances

  • Interleukin-1
  • Interleukin-6
  • Oligodeoxyribonucleotides
  • Oligonucleotides, Antisense
  • RNA, Messenger
  • Receptors, Interleukin-1
  • Dinoprostone