Chronic prenatal and postnatal Pb2+ exposure increases [3H]MK801 binding sites in adult rat forebrain

Eur J Pharmacol. 1993 Oct 1;248(3):273-5. doi: 10.1016/0926-6917(93)90054-t.

Abstract

We have measured the binding of [3H]MK801 to the N-methyl-D-aspartate (NMDA) receptor-ion channel in membrane preparations from adult rat forebrain exposed to lead (Pb2+) during gestation, lactation, and postweaning. Our results indicate a 30.9% increase in the number of [3H]MK801 binding sites in Pb2+ exposed animals relative to controls. No significant changes in the affinity constant were observed. The level of blood Pb2+ for which such changes were measured was 13.9 +/- 2.8 micrograms/dl. These results indicate that alterations in the NMDA receptor-ion channel complex are present at blood Pb2+ levels which are environmentally relevant and suggest that chronic Pb2+ exposure during development can influence the NMDA receptor complex in the adult rat brain.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Animals, Newborn / blood
  • Animals, Newborn / growth & development
  • Binding Sites / drug effects
  • Dizocilpine Maleate / pharmacokinetics*
  • Female
  • Lead / blood
  • Lead / toxicity*
  • Pregnancy
  • Prenatal Exposure Delayed Effects*
  • Prosencephalon / drug effects*
  • Prosencephalon / metabolism
  • Rats
  • Receptors, N-Methyl-D-Aspartate / drug effects*

Substances

  • Receptors, N-Methyl-D-Aspartate
  • Lead
  • Dizocilpine Maleate