Possible involvement of protein kinase C activation in down-regulation of CD3 antigen on adult T cell leukaemia cells

Br J Haematol. 1994 Feb;86(2):399-401. doi: 10.1111/j.1365-2141.1994.tb04751.x.

Abstract

The role of protein kinase C (PKC) system on CD3 expression on adult T-cell leukaemia (ATL) was examined. The down-regulation of CD3 on ATL cells is reportedly induced by CD3 down-regulating factor (CD3DF) contained in serum and culture supernatants of leukaemia cells from acute type ATL patients. After we cultured normal PBMC with a PKC inhibitor, H-7, CD3DF activity for PBMC was reduced significantly. Culture with H-7 of HTLV-1 transformed T cells, ATL-2 cells whose CD3 expression had been decreased, led to enhancement of CD3 expression in a time-dependent manner. These findings suggest that CD3DF may play an important role as a PKC system activator, resulting in CD3 down-regulation.

MeSH terms

  • 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine
  • Antigens, Neoplasm / analysis*
  • Antigens, Neoplasm / drug effects
  • CD3 Complex / analysis*
  • CD3 Complex / drug effects
  • Down-Regulation / physiology*
  • Humans
  • Isoquinolines / pharmacology
  • Leukemia-Lymphoma, Adult T-Cell / enzymology
  • Leukemia-Lymphoma, Adult T-Cell / immunology*
  • Piperazines / pharmacology
  • Protein Kinase C / antagonists & inhibitors
  • Protein Kinase C / physiology*
  • Tumor Cells, Cultured

Substances

  • Antigens, Neoplasm
  • CD3 Complex
  • Isoquinolines
  • Piperazines
  • 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine
  • Protein Kinase C