Near-visible-UV radiation delays UVB tumorigenesis

J Photochem Photobiol B. 1994 Feb;22(2):119-23. doi: 10.1016/1011-1344(93)06956-4.

Abstract

The effect of UVA radiation (321-400 nm) on UVB photocarcinogenesis was examined in lightly pigmented hairless hr/hr C3H/Tif mice. Five groups of 22 mice were exposed to UVB radiation (281-320 nm) from one Philips 12 tube for 10 min per day and 4 days per week. Four of the groups were simultaneously exposed to UVA from two to six filtered Philips 09 tubes. The daily dose of UVB was 2.0 kJ m-2 in all five groups; the UVA2 (321-340 nm) dose varied from 0.7 to 4.5 kJ m-2 and the UVA1 (341-400 nm) dose from 0.3 to 45.6 kJ m-2. A sixth group was exclusively irradiated with the filtered UVA tubes and served as a control. Skin tumor development was not significantly different for the groups exposed to the UVB source alone or the UVB source in combination with the largest daily UVA dose (0.2 > p > 0.1). Skin tumor development was significantly delayed in the other groups irradiated with the UVB source and the lower doses of UVA (p < 0.001). No tumors were observed in the control group. This study suggests that UVA1 radiation delays UVB-induced skin tumor development. However, the delay cannot be expected to persist when UVA is administered in higher daily doses.

Publication types

  • Comparative Study

MeSH terms

  • Animals
  • Dose-Response Relationship, Radiation
  • Female
  • Mice
  • Mice, Hairless
  • Mice, Inbred C3H
  • Neoplasms, Radiation-Induced / etiology
  • Neoplasms, Radiation-Induced / pathology*
  • Skin / radiation effects*
  • Skin Neoplasms / etiology
  • Skin Neoplasms / pathology*
  • Time Factors
  • Ultraviolet Rays*