Abstract
NuMA is a protein involved in maintenance of nuclear structure and in the assembly of the mitotic spindle. Expression of amino-terminal deletion mutants results in a phenotype identical to that caused by a temperature-sensitive defect of RCC1 (regulator of chromosome condensation). Here we describe the isolation of NuMA protein from HeLa cells under mild conditions as a prerequisite to study its interactions with elements of the RCC1-Ran regulatory pathway. In an overlay assay, NuMA did not bind Ran.[gamma-32P]GTP. Thus it is clearly different from Ran.GTP binding proteins of similar M(r).
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Antigens, Nuclear
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Cell Cycle Proteins
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Chromatography, Gel
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Fluorescent Antibody Technique
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GTP-Binding Proteins
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Guanosine Triphosphate / metabolism
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HeLa Cells / chemistry
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Humans
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Immunoblotting
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Interphase
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Mitosis
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Nuclear Matrix-Associated Proteins
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Nuclear Proteins / isolation & purification*
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Nuclear Proteins / metabolism
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Spindle Apparatus*
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ran GTP-Binding Protein
Substances
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Antigens, Nuclear
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Cell Cycle Proteins
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NUMA1 protein, human
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Nuclear Matrix-Associated Proteins
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Nuclear Proteins
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Guanosine Triphosphate
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GTP-Binding Proteins
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ran GTP-Binding Protein