Phosphorylation of calmodulin by the epidermal-growth-factor-receptor tyrosine kinase

Eur J Biochem. 1994 Sep 15;224(3):909-16. doi: 10.1111/j.1432-1033.1994.00909.x.

Abstract

An epidermal-growth-factor(EGF)-receptor preparation isolated by calmodulin-affinity chromatography from rat liver plasma membranes is able to phosphorylate calmodulin. Calmodulin phosphorylation was enhanced 3-8-fold by EGF, was dependent on the presence of a polycation or basic protein and was inhibited by micromolar concentrations of Ca2+. Phosphate incorporation into calmodulin occurs predominantly on tyrosine residues. Partial proteolysis of phosphocalmodulin by thrombin identifies Tyr99, located in the third calcium-binding domain of calmodulin, as the phosphorylated residue. Stoichiometric measurements show a 32P/calmodulin molar ratio of approximately 1 when optimal phosphorylation conditions are used.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Calmodulin / metabolism*
  • Electrophoresis, Polyacrylamide Gel
  • ErbB Receptors / metabolism*
  • Kinetics
  • Liver / metabolism
  • Male
  • Phosphorylation
  • Rats
  • Rats, Sprague-Dawley

Substances

  • Calmodulin
  • ErbB Receptors