Effect of glutathione depletion and oral N-acetyl-cysteine treatment on CD4+ and CD8+ cells

FASEB J. 1994 Apr 1;8(6):448-51.

Abstract

HIV-infected individuals and SIV-infected rhesus macaques have, on the average, decreased plasma cysteine and cystine concentrations and decreased intracellular glutathione levels. We show that the cysteine supply and the intracellular glutathione levels have a strong influence on the T cell system. A study of healthy human subjects revealed that persons with intracellular glutathione levels of 20-30 nmol/mg protein had significantly higher numbers of CD4+ T cells than persons with either lower or higher glutathione levels. Persons who moved during a 4-week observation period from the optimal to the suboptimal range (10-20 nmol/mg) experienced, on the average, a 30% decrease in CD4+ T cell numbers. This decrease was prevented by treatment with N-acetyl-cysteine (NAC). NAC caused this relative increase of CD4+ T cell numbers in spite of decreasing glutathione levels and not by increasing the glutathione level. Our studies suggest that the immune system may be exquisitely sensitive not only against a cysteine and glutathione deficiency but also against an excess of cysteine.

Publication types

  • Clinical Trial
  • Randomized Controlled Trial

MeSH terms

  • Acetylcysteine / administration & dosage
  • Acetylcysteine / pharmacology*
  • Administration, Oral
  • Adult
  • CD4-Positive T-Lymphocytes / drug effects*
  • CD8 Antigens / analysis*
  • Double-Blind Method
  • Glutathione / analysis*
  • Humans
  • Leukocyte Count / drug effects
  • Male
  • Middle Aged
  • T-Lymphocytes / drug effects*

Substances

  • CD8 Antigens
  • Glutathione
  • Acetylcysteine