Effect of extracellular Mg2+ concentration on agonist-induced cytosolic free Ca2+ transients

Biochim Biophys Acta. 1995 Feb 16;1265(1):89-92. doi: 10.1016/0167-4889(94)00231-3.

Abstract

The modulating effects of extracellular Mg2+ concentration ([Mg2+]o) on vascular contraction are well known. In the present study it was tested how the changes in cytosolic free Ca2+ concentration ([Ca2+]i) induced by various agonists are modified by changes in [Mg2+]o. Extracellular Mg2+ deprivation increased the [Ca2+]i response to arginine vasopressin, to angiotensin II and to thapsigargin, but not to 5-hydroxytryptamine and noradrenaline. Withdrawal of extracellular Ca2+ revealed that extracellular Mg2+ deprivation increased Ca2+ influx, but not Ca2+ release from cellular stores. The findings demonstrate that different responses of [Ca2+]i to agonists may underlie the modulating effect of [Mg2+]o on vascular contraction.

MeSH terms

  • Angiotensin II / pharmacology
  • Animals
  • Arginine Vasopressin / pharmacology
  • Calcium / analysis*
  • Cells, Cultured
  • Cytosol / metabolism
  • Dose-Response Relationship, Drug
  • Fura-2
  • Magnesium / pharmacology*
  • Male
  • Muscle, Smooth, Vascular / drug effects
  • Muscle, Smooth, Vascular / metabolism*
  • Rats
  • Rats, Inbred WKY
  • Terpenes / pharmacology
  • Thapsigargin

Substances

  • Terpenes
  • Angiotensin II
  • Arginine Vasopressin
  • Thapsigargin
  • Magnesium
  • Calcium
  • Fura-2