Transforming growth factor-beta: antisense RNA-mediated inhibition affects anchorage-independent growth, tumorigenicity and tumor-infiltrating T-cells in malignant mesothelioma

Growth Factors. 1994;11(1):29-44. doi: 10.3109/08977199409015049.

Abstract

Transforming growth factor-beta (TGF-beta) is produced by a number of tumor cell types including human malignant mesothelioma (MM), but its role as a direct or indirect factor in tumorigenesis is incompletely understood. We have investigated the expression of TGF-beta isoforms by human and murine MM cells and have analysed the effects of inducible antisense RNA-mediated inhibition of TGF-beta expression on murine MM in vitro and in vivo. The results showed that (a) TGF-beta 1 and -beta 2 were produced by both human and mouse MM cells, (b) antisense RNA against either TGF-beta 1 or -beta 2 cross-inhibited both TGF-beta 1 and -beta 2 expression, (c) inhibition of TGF-beta expression reduced the anchorage-independent growth of MM cells in vitro and the tumorigenicity of MM cells in vivo, and (d) inhibition of TGF-beta expression led to increased T lymphocyte infiltration into tumors. The data suggest that TGF-beta has multiple tumor-enhancing effects in MM.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Base Sequence
  • Cell Adhesion / physiology
  • Cell Division / physiology
  • DNA Primers / genetics
  • Female
  • Genetic Vectors
  • Humans
  • Lymphocytes, Tumor-Infiltrating / pathology
  • Mesothelioma / etiology*
  • Mesothelioma / pathology
  • Mice
  • Mice, Inbred CBA
  • Molecular Sequence Data
  • Polymerase Chain Reaction
  • RNA, Antisense / genetics
  • RNA, Antisense / pharmacology
  • RNA, Messenger / genetics
  • Transforming Growth Factor beta / antagonists & inhibitors*
  • Transforming Growth Factor beta / genetics
  • Transforming Growth Factor beta / physiology
  • Tumor Cells, Cultured / pathology

Substances

  • DNA Primers
  • RNA, Antisense
  • RNA, Messenger
  • Transforming Growth Factor beta