Mechanisms in adverse reactions to food. The eye

Allergy. 1995;50(20 Suppl):68-73.

Abstract

Direct action of triggers on the target organ may cause vasodilation, exudation, hypersecretion and nerve endings stimulation without participation of mast cells and basophils. Non IgE-mediated release of mediators from mast cells and basophils may explain how triggers other than allergens can induce pseudo-allergic reactions. IgE-mediated triggering of mast cells and basophils is not confined - in the eye too - to an immediate reaction, but represents the starting point for a progression to a late-phase reaction with the participation of a network of several cells and mediators. Inflammatory changes associated with late-phase reaction in the eye represent the pathophysiological basis for a non-specific hyperreactivity to various triggers.

MeSH terms

  • Conjunctivitis, Allergic / diagnosis
  • Conjunctivitis, Allergic / etiology*
  • Conjunctivitis, Allergic / physiopathology
  • Eye / physiopathology
  • Food Hypersensitivity / complications*
  • Food Hypersensitivity / immunology
  • Food Hypersensitivity / physiopathology
  • Humans
  • Immunoglobulin E / metabolism

Substances

  • Immunoglobulin E