Pretreatment with nimodipine prevents MPTP-induced neurotoxicity at the nigral, but not at the striatal level in mice

Neuroreport. 1995 Mar 7;6(4):621-5. doi: 10.1097/00001756-199503000-00009.

Abstract

The present study assessed the effects of pretreatment with the calcium-L-type channel blocker nimodipine on biochemical and histological parameters of systemic MPTP-induced (2 x 40 mg kg-1 body weight subcutaneously, 16 h apart), dopaminergic neurotoxicity in black C57BL/6 mice. Continuous administration of nimodipine via subcutaneously implanted pellets (10 mg), starting 7 days before administration of MPTP, did not antagonize the striatal MPTP-induced dopamine depletion (caudate-putamen: 80% of control; nucleus accumbens; 25% of control), but almost completely prevented the MPTP-induced tyrosine hydroxylase immunoreactive-cell loss in the substantia nigra (20% of control) 7 days after administration of MPTP. This data suggests that pretreatment with nimodipine--during the observation period of 7 days--protects against MPTP-induced neurotoxicity in mice at the nigral ('cell body'), but not at the synaptic striatal level.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine / antagonists & inhibitors*
  • Animals
  • Biogenic Monoamines / metabolism
  • Corpus Striatum / drug effects*
  • Corpus Striatum / metabolism
  • Female
  • Linear Models
  • Mice
  • Mice, Inbred C57BL
  • Nimodipine / pharmacology*
  • Substantia Nigra / drug effects*
  • Substantia Nigra / metabolism

Substances

  • Biogenic Monoamines
  • Nimodipine
  • 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine