Abstract
Long-term exposure of platelets to prostacyclin or iloprost (100nM, 3hr) results in receptor desensitization measured as decrease in 3H-iloprost binding sites by 47 +/- 14%. Desensitized platelets respond with an increased adhesion to endothelial cells. The mechanism of increased adhesiveness was studied by measuring the expression of the adhesion molecule CD62p (p-selectin; GMP140) on washed human platelets by flowcytometry. In thrombin stimulated platelets CD62p expression was dose-dependently reduced by iloprost. In receptor desensitized platelets IC50 for iloprost inhibition of thrombin-induced CD62p expression increased from 0.48 +/- 0.10 to 2.4 +/- 0.7 nM.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Analysis of Variance
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Blood Platelets / drug effects
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Blood Platelets / metabolism*
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Cell Adhesion Molecules / biosynthesis*
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Cells, Cultured
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Endothelium, Vascular / cytology
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Endothelium, Vascular / drug effects
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Endothelium, Vascular / metabolism
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Flow Cytometry
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Fluorescent Antibody Technique
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Humans
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Iloprost / metabolism*
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Iloprost / pharmacology
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P-Selectin
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Platelet Membrane Glycoproteins / biosynthesis*
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Receptors, Cell Surface / drug effects
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Receptors, Cell Surface / metabolism*
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Receptors, Epoprostenol
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Receptors, Prostaglandin / drug effects
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Receptors, Prostaglandin / metabolism*
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Thrombin / pharmacology
Substances
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Cell Adhesion Molecules
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P-Selectin
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Platelet Membrane Glycoproteins
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Receptors, Cell Surface
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Receptors, Epoprostenol
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Receptors, Prostaglandin
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Thrombin
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Iloprost