Induction of heme oxygenase and inhibition of delta-aminolevulinic acid synthetase of rat liver by thioacetamide and thioacetamide-S-oxide

J Pharmacobiodyn. 1983 May;6(5):340-5. doi: 10.1248/bpb1978.6.340.

Abstract

Thioacetamide and one of its metabolite, thioacetamide-S-oxide, were shown to increase heme oxygenase and to inhibit delta-aminolevulinic acid (ALA) synthetase when administered in vivo to male rats. Concomitant with the increase of heme oxygenase and the decrease of ALA synthetase, concentration of cytochrome P-450 and drug metabolizing enzyme activities were decreased by in vivo administration of thioacetamide and thioacetamide-S-oxide to rats. The results of these studies indicate that thioacetamide and thioacetamide-S-oxide are not only inhibitor of ALA synthetase, but also inducer of heme oxygenase in rats. Further, thioacetamide-S-oxide is generally more effective than thioacetamide with respect to the effects on cytochrome P-450, ALA synthetase and heme oxygenase.

MeSH terms

  • 5-Aminolevulinate Synthetase / antagonists & inhibitors*
  • Acetamides / pharmacology*
  • Animals
  • Cytochrome P-450 Enzyme System / metabolism
  • Enzyme Induction / drug effects
  • Heme Oxygenase (Decyclizing) / biosynthesis*
  • Liver / enzymology*
  • Male
  • Mixed Function Oxygenases / biosynthesis*
  • Rats
  • Rats, Inbred Strains
  • Thioacetamide / analogs & derivatives
  • Thioacetamide / pharmacology*
  • Time Factors

Substances

  • Acetamides
  • Thioacetamide
  • thioacetamide-S-oxide
  • Cytochrome P-450 Enzyme System
  • Mixed Function Oxygenases
  • Heme Oxygenase (Decyclizing)
  • 5-Aminolevulinate Synthetase