Effect of dexamethasone on nuclear envelope nucleoside triphosphatase in fibroblasts 3T3 and melanoma cells MMLI

J Invest Dermatol. 1984 Jul;83(1):20-2. doi: 10.1111/1523-1747.ep12261641.

Abstract

Glucocorticoids are known to influence DNA and RNA synthesis in skin fibroblasts. In a novel approach to study this effect, we investigated the influence of the hormone analogue dexamethasone on the activity of nuclear envelope-associated nucleoside triphosphatase (NTPase) in intact cell systems (3T3 fibroblasts and MMLI melanoma cells). The NTPase is thought to be responsible for regulation of nucleocytoplasmic transport of mRNA. [3H]Dexamethasone was found to bind to nuclear ghosts at a density comparable with that of nuclear pores in this cellular fraction. Incubation of the cells for 48 h in the presence of different concentrations of dexamethasone resulted in a marked decrease of NTPase activity. Already concentrations as low as 0.1 ng/ml (3T3) or 1 ng/ml (MMLI) reduced the NTPase activity by approximately 50%. These results suggest that nuclear envelope NTPase is a site at which glucocorticoids regulate gene expression.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Biological Transport / drug effects
  • Cell Line
  • Cells, Cultured
  • Dexamethasone / metabolism
  • Dexamethasone / pharmacology*
  • Dose-Response Relationship, Drug
  • Fibroblasts / drug effects
  • Fibroblasts / enzymology
  • Humans
  • Melanoma / enzymology*
  • Mice
  • Nuclear Envelope / drug effects*
  • Nuclear Envelope / enzymology
  • Nucleoside-Triphosphatase
  • Phosphoric Monoester Hydrolases / metabolism*
  • RNA, Messenger / metabolism
  • RNA, Neoplasm / metabolism
  • Time Factors

Substances

  • RNA, Messenger
  • RNA, Neoplasm
  • Dexamethasone
  • Phosphoric Monoester Hydrolases
  • Nucleoside-Triphosphatase