Erythromycin toxicity in primary cultures of rat hepatocytes

Xenobiotica. 1985 Aug-Sep;15(8-9):767-73. doi: 10.3109/00498258509047439.

Abstract

Cultured rat hepatocytes were used to study the toxicity of erythromycin base (EB), erythromycin estolate (EE) and a new fluorinated derivative, (8S)-8-fluoroerythromycin A (EF). EF was not cytotoxic after 18 h incubation at concentrations up to 8 X 10(-4) M and EE was much more toxic than EB at all concentrations studied. EE toxicity was greater in a serum-free medium and was not increased by induction of cytochrome P-450 with phenobarbitone. In hepatocytes co-cultured with rat-liver epithelial cells EE, but not EF, raised the cytochrome P-450 content and formed stable cytochrome P-450 complexes with about 40% of the haemoprotein. The lack of correlation between cytochrome P-450 content and cytotoxicity suggests that some of the parent erythromycin drugs and not their metabolites are the toxic entities.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Survival / drug effects
  • Cells, Cultured
  • Cytochrome P-450 Enzyme System / metabolism
  • Erythromycin / analogs & derivatives
  • Erythromycin / toxicity*
  • Erythromycin Estolate / toxicity
  • L-Lactate Dehydrogenase / metabolism
  • Liver / drug effects*
  • Liver / metabolism
  • Liver / pathology
  • Male
  • Rats
  • Rats, Inbred Strains

Substances

  • flurithromycin
  • Erythromycin
  • Cytochrome P-450 Enzyme System
  • L-Lactate Dehydrogenase
  • Erythromycin Estolate