Pharmacological characterization of alpha adrenoceptor-mediated motor responses in the rat colon

Neurogastroenterol Motil. 2025 Jan;37(1):e14921. doi: 10.1111/nmo.14921. Epub 2024 Sep 30.

Abstract

Background: Inhibitory neuromuscular transmission in the gastrointestinal tract is mediated by intrinsic nitrergic and purinergic neurons. Purines activate G protein-coupled receptor P2Y1 receptors, increasing intracellular Ca2+ that activates small conductance calcium-activated potassium (SKCa) channels. Little is known about the effect of adrenergic receptor activation on intestinal smooth muscle. In vascular tissue, stimulation of α-adrenoceptors causes smooth muscle contraction, while their effect on intestinal tissue is poorly understood. This study aimed to pharmacologically characterize the effect of α-adrenoceptor activation in the rat colon, which shares similar inhibitory pathways to the human colon.

Methods: Muscle bath experiments were performed with the rat proximal, mid, and distal colon oriented both circularly and longitudinally.

Results: The α1-adrenoceptor agonist phenylephrine (PE) (10-8-10-5 M) evoked concentration-dependent relaxations of the intestinal smooth muscle from all regions and orientations. However, in the mid-circular colon at low PE concentrations, a contraction sensitive to 10-5 M phentolamine (non-selective α-adrenoceptor blocker), the neural blocker tetrodotoxin (TTX; 10-6 M), and atropine (10-6 M) was recorded. PE-induced relaxations were insensitive to TTX (10-6 M) and the nonselective β-adrenoceptor blocker propranolol (10-6 M). In contrast, PE-induced relaxations were blocked by phentolamine (10-5 M), prazosin (10-6 M) (α1-adrenoceptor blocker), and RS17053 (10-6 M) (α1A-blocker), but not by yohimbine (10-6 M) (α2-adrenoceptor blocker). Apamin (10-6 M), a SKCa channel blocker, abolished PE-induced relaxations.

Conclusions: Contractile responses in the circular muscle of the mid colon could be attributed to α-adrenoceptors located on enteric cholinergic neurons. Stimulation of α1A-adrenoreceptors activates SKCa channels to cause smooth muscle relaxation, which constitutes a signaling pathway that shares similarities with P2Y1 receptors.

Keywords: alpha adrenoceptors; apamin; colon; phenylephrine; relaxation.

MeSH terms

  • Adrenergic alpha-1 Receptor Agonists / pharmacology
  • Adrenergic alpha-Antagonists / pharmacology
  • Animals
  • Colon* / drug effects
  • Colon* / metabolism
  • Colon* / physiology
  • Gastrointestinal Motility / drug effects
  • Gastrointestinal Motility / physiology
  • Male
  • Muscle Contraction* / drug effects
  • Muscle Contraction* / physiology
  • Muscle, Smooth* / drug effects
  • Muscle, Smooth* / metabolism
  • Muscle, Smooth* / physiology
  • Phenylephrine / pharmacology
  • Rats
  • Rats, Sprague-Dawley
  • Rats, Wistar
  • Receptors, Adrenergic, alpha / drug effects
  • Receptors, Adrenergic, alpha / metabolism
  • Receptors, Adrenergic, alpha / physiology

Substances

  • Receptors, Adrenergic, alpha
  • Phenylephrine
  • Adrenergic alpha-Antagonists
  • Adrenergic alpha-1 Receptor Agonists