Depression and risk of arthritis: A Mendelian randomization study

Brain Behav. 2024 Jun;14(6):e3551. doi: 10.1002/brb3.3551.

Abstract

Introduction: Observational studies have found that most patients with arthritis have depression. We aimed to determine the causal relationship between various types of arthritis and depression.

Methods: We conducted a two-sample bidirectional Mendelian randomized (MR) analysis to determine whether there was a significant causal relationship between depression and multiple types of arthritis. The data of our study were derived from the publicly released genome-wide association studies (GWASs) and the largest GWAS meta-analysis. MR analysis mainly used inverse-variance weighted method; supplementary methods included weighted median, weighted mode, and MR-Egger using MR pleiotropy residual sum and outlier to detect and correct for the presence of pleiotropy.

Results: After adjusting for heterogeneity and horizontal pleiotropy, we found that depression was associated with an increased risk of osteoarthritis (OA) (OR = 1.02, 95%CI: 1.01-1.02, p = 2.96 × E - 5). In the reverse analysis, OA was also found to increase the risk of depression (OR = 1.10, 95%CI: 1.04-1.15, p = .0002). Depression only increased the risk of knee OA (KOA) (OR = 1.25, 95%CI: 1.10-1.42, p = 6.46 × E - 4). Depression could potentially increase the risk of spondyloarthritis (OR = 1.52, 95%CI: 1.19-1.94, p ≤ 8.94 × E - 4).

Conclusion: There is a bidirectional causal relationship of depression with OA. However, depression only augments the risk of developing KOA. Depression may increase the risk of spondyloarthritis and gout.

Keywords: Mendelian randomization; arthritis; depressive.

MeSH terms

  • Arthritis / epidemiology
  • Arthritis / genetics
  • Arthritis, Rheumatoid / epidemiology
  • Arthritis, Rheumatoid / genetics
  • Depression* / epidemiology
  • Depression* / genetics
  • Genome-Wide Association Study*
  • Gout / epidemiology
  • Gout / genetics
  • Humans
  • Mendelian Randomization Analysis* / methods
  • Osteoarthritis* / epidemiology
  • Osteoarthritis* / genetics
  • Osteoarthritis, Knee / epidemiology
  • Osteoarthritis, Knee / genetics
  • Risk Factors
  • Spondylarthritis / genetics